Copyright
©The Author(s) 2022.
World J Clin Oncol. Mar 24, 2022; 13(3): 186-199
Published online Mar 24, 2022. doi: 10.5306/wjco.v13.i3.186
Published online Mar 24, 2022. doi: 10.5306/wjco.v13.i3.186
Figure 1 Host immune response to age-stratified Helicobacter pylori infection.
H. pylori: Helicobacter pylori; IL: Interleukin; TNF-α: Tumor necrosis factor α; IFN-γ: Interferon γ; T Reg Cells: Regulatory T cells.
Figure 2 Summary scheme on the microenvironment of gastric cancer.
TGF-β: Transforming growth factor beta; FGF5: Fibroblast growth factor 5; GAS6: Specific growth arrest protein 6; VEGF: Vascular endothelial growth factor; CXCL9: Chemokine ligand; PD-L1: Programmed death ligand 1; CTLA-4: Cytotoxic T-lymphocyte-associated protein 4; IL: Interleukin; Th: T helper cell; TNF: Tumor necrosis factor; Treg: Regulatory T cell.
- Citation: Lima de Souza Gonçalves V, Cordeiro Santos ML, Silva Luz M, Santos Marques H, de Brito BB, França da Silva FA, Souza CL, Oliveira MV, de Melo FF. From Helicobacter pylori infection to gastric cancer: Current evidence on the immune response. World J Clin Oncol 2022; 13(3): 186-199
- URL: https://www.wjgnet.com/2218-4333/full/v13/i3/186.htm
- DOI: https://dx.doi.org/10.5306/wjco.v13.i3.186