Published online Jun 24, 2019. doi: 10.5306/wjco.v10.i6.234
Revised: March 31, 2019
Accepted: April 8, 2019
Published online: June 24, 2019
Processing time: 155 Days and 15.6 Hours
SOX2 is a significant regulator of pluripotent cellular transcription and it helps in the reprogramming of somatic cells to the pluripotent properties. The involvement of SOX2 in cancer biology has been recently demonstrated.
Metastasis comprises the most important aspect of cancer-related mortality. Linking SOX2 expression to metastasis and subsequently to patient’s survival may open novel horizons for the implementation of future therapeutic strategies that target SOX2 biological pathways.
To investigate the association between SOX2 overexpression and the development of a metastatic phenotype as well as the survival patterns of patients with increased SOX2 ex-pression.
A meta-analysis was conducted, including studies that recruited patients with different types of cancer and reporting SOX2 expression as either “low” or “high”, and evaluating patient survival using the relevant analytical methods [overall survival (OS) and/or disease-free survival (DFS)]. A comprehensive search of articles published between 2010 and 2018 was performed in distinct scientific databases.
A total of 20 studies involving 2643 patients (60.88% males) were included. SOX2 overexpression was significantly associated with distant metastasis (odds ratio = 1.79, 95%CI: 1.20-3.25, P < 0.008), while it was associated with lymph node metastasis only in subgroup analyses of cancers of the colon, breast, and lung. Both OS and DFS were shorter in patients expressing high SOX2, as compared to those with low SOX2 expression (hazard ratio = 1.65, 95%CI: 1.34-2.04, P < 0.001 and hazard ratio = 1.54, 95%CI: 1.14-2.08, P = 0.005, respectively).
The present study adds a comprehensive insight into the significant role of SOX2 in distant metastasis of different types of cancers and its correlation to poor prognosis rather than the outcomes obtained by individual studies. In line with the increased research interest in SOX2, we showed that it can be used as a prognostic marker in cancer patients, while, on the other hand, new therapeutic strategies are urgently needed to target the biological pathways implicated in SOX2 overexpression for more effective cancer treatment.
Targeting SOX2 expression in cancer regimens is warranted. Future research studies should focus on developing novel drugs as well as the identification of the cut-off values of poor prognosis.