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World J Clin Oncol. Dec 10, 2011; 2(12): 384-396
Published online Dec 10, 2011. doi: 10.5306/wjco.v2.i12.384
IL-6/IL-6R as a potential key signaling pathway in prostate cancer development
Andreia Azevedo, Virginia Cunha, Ana Luisa Teixeira, Rui Medeiros
Andreia Azevedo, Virginia Cunha, Ana Luisa Teixeira, Rui Medeiros, Molecular Oncology and Virology, Portuguese Institute of Oncology, 4200-072 Porto, Portugal
Andreia Azevedo, Rui Medeiros, Research Department of Portuguese League against Cancer (NRNorte), 4200-072 Porto, Portugal
Rui Medeiros, CEBIMED, Faculty of Health Sciences of Fernando Pessoa University, 4200-150 Porto, Portugal
Ana Luisa Teixeira, Rui Medeiros, ICBAS, Abel Salazar Institute for the Biomedical Sciences, University of Porto, 4200-072 Porto, Portugal
Author contributions: Azevedo A and Cunha V designed the structure of the review; Azevedo A wrote the initial draft and the final version of the manuscript; Cunha V, Teixeira AL and Medeiros R critically revised the manuscript for important intellectual content; Medeiros R supervised the study and approved the version to be published.
Supported by Calouste Gulbenkian Foundation (Oncology/2008/Project n 96736) and Science and Technology Foundation (FCT/PTDC/SAU-FCF/71552/2006)
Correspondence to: Rui Medeiros, Professor, Molecular Oncology and Virology, Portuguese Institute of Oncology, Porto, R. Dr. António Bernardino de Almeida, 4200-072 Porto, Portugal. ruimedei@ipoporto.min-saude.pt
Telephone: +351-22-5084000 Fax: +351-22-5084001
Received: September 9, 2011
Revised: November 8, 2011
Accepted: November 15, 2011
Published online: December 10, 2011
Abstract

Interleukin-6 (IL-6) is a pleiotropic cytokine involved in prostate regulation and in prostate cancer (PC) development/progression. IL-6 acts as a paracrine and autocrine growth stimulator in benign and tumor prostate cells. The levels of IL-6 and respective receptors are increased during prostate carcinogenesis and tumor progression. Several studies reported that increased serum and plasma IL-6 and soluble interleukin-6 receptor levels are associated with aggressiveness of the disease and are associated with a poor prognosis in PC patients. In PC treatment, patients diagnosed with advanced stages are frequently submitted to hormonal castration, although most patients will eventually fail this therapy and die from recurrent castration-resistant prostate cancer (CRPC). Therefore, it is important to understand the mechanisms involved in CRPC. Several pathways have been proposed to be involved in CRPC development, and their understanding will improve the way to more effective therapies. In fact, the prostate is known to be dependent, not exclusively, on androgens, but also on growth factors and cytokines. The signaling pathway mediated by IL-6 may be an alternative pathway in the CRPC phenotype acquisition and cancer progression, under androgen deprivation conditions. The principal goal of this review is to evaluate the role of IL-6 pathway signaling in human PC development and progression and discuss the interaction of this pathway with the androgen recepto pathway. Furthermore, we intend to evaluate the inclusion of IL-6 and its receptor levels as a putative new class of tumor biomarkers.The IL-6/IL-6R signaling pathway may be included as a putative molecular marker for aggressiveness in PC and it may be able to maintain tumor growth through the AR pathway under androgen-deprivation conditions. The importance of the IL-6/IL-6R pathway in regulation of PC cells makes it a good candidate for targeted therapy.

Keywords: Androgen receptor; Castration-resistant prostate cancer; Hormonal castration; Inteuleukin-6; Inteuleukin-6 receptor; Prostate cancer; Tumor biomarker