Current understanding of the neuropathophysiology of pain in chronic pancreatitis

doi:10.4291/wjgp.v6.i4.193

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World Journal of Gastrointestinal Pathophysiology

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2150-5330

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastrointest Pathophysiol. Nov 15, 2015; 6(4): 193-202
Published online Nov 15, 2015. doi: 10.4291/wjgp.v6.i4.193

Table 1 Potential treatment alternatives and their drug targets

Drug target	Potential treatment alternatives
NGF	Tanezumab
TRPV1	TRPV1 antagonist
PAR2	Trypsin inhibitors
Mast cell	Ketotifen
Interleukin 1	Recombinant interleukin-1 receptor antagonist
Interleukin 6	Interleukin-6 antagonist
Central sensitization	Ketamine, dextromethrophan, pregabalin, tricyclic antidepressants, and noradrenaline reuptake inhibitors

NGF: Nerve growth factor; TRPV1: Transient receptor potential vanilloid 1; PAR2: Proteinase-activated receptor 2.

Citation: Atsawarungruangkit A, Pongprasobchai S. Current understanding of the neuropathophysiology of pain in chronic pancreatitis. World J Gastrointest Pathophysiol 2015; 6(4): 193-202
URL: https://www.wjgnet.com/2150-5330/full/v6/i4/193.htm
DOI: https://dx.doi.org/10.4291/wjgp.v6.i4.193