Published online Nov 15, 2015. doi: 10.4291/wjgp.v6.i4.193
Peer-review started: April 12, 2015
First decision: July 10, 2015
Revised: July 22, 2015
Accepted: September 10, 2015
Article in press: September 16, 2015
Published online: November 15, 2015
Processing time: 220 Days and 20.8 Hours
Chronic pancreatitis (CP) is a chronic inflammatory disease of the pancreas. The main symptom of patients with CP is chronic and severe abdominal pain. However, the pathophysiology of pain in CP remains obscure. Traditionally, researchers believed that the pain was caused by anatomical changes in pancreatic structure. However, treatment outcomes based on such beliefs are considered unsatisfactory. The emerging explanations of pain in CP are trending toward neurobiological theories. This article aims to review current evidence regarding the neuropathophysiology of pain in CP and its potential implications for the development of new treatments for pain in CP.
Core tip: Abdominal pain is the main symptom of patients with chronic pancreatitis (CP), yet the underlying mechanisms are not well understood. The emerging explanations of pain in CP are trending toward neurobiological theories. This article reviews these emerging concepts and their potential implications for the development of new treatments for pain in CP. Three major concepts attempting to explain the pathogenesis of CP pain: Pancreatic nociception and sensitization-induced pain, neuropathic remodeling, and central mechanism of pancreatitis pain are summarized, along with the specific molecules involved in each and potential therapeutic targets.