Published online Jan 26, 2019. doi: 10.4330/wjc.v11.i1.24
Peer-review started: September 19, 2018
First decision: October 26, 2018
Revised: December 16, 2018
Accepted: December 24, 2018
Article in press: December 24, 2018
Published online: January 26, 2019
Processing time: 129 Days and 8.6 Hours
Obesity is independently associated with mortality and cardiovascular disease. However, once cardiovascular disease is established, the studies published so far show contradictory results. On the other hand, several epidemiological studies have demonstrated an independent association of carotid disease, defined as carotid plaques or carotid intima media thickness, with overall mortality and cardiovascular events.
There are no studies addressing the value of subclinical atherosclerosis, defined as carotid disease, and adverse events in obese patients with or without cardiovascular disease.
This study aimed to determine if carotid disease is a predictor of adverse events in obese patients with coronary artery disease suspicion and negative treadmill stress echocardiography.
A retrospective cohort study of patients without significant heart or vascular disease, body mass index ≥ 30 kg/m2 and coronary artery disease suspicion with negative exercise echocardiography (EE) submitted to carotid ultrasonography. Between January 2006 and December 2010, 2000 patients were submitted for stress echocardiography and carotid ultrasonography in our centre. Of them, 226 (11.3%) were included. Adverse events were defined as all-cause mortality, myocardial infarction and cerebrovascular accident.
We found that 226 patients had body mass indexes ≥ 30 kg/m2, and 76 of them (33.6%) had carotid plaques. During a mean follow-up time of 8.2 (2.1) years, 27 adverse events were found (11.9%). Mean event-free survival at 1, 5 and 10 years was 99.1% (0.6), 95.1% (1.4) and 86.5% (2.7), respectively. In univariate analysis, carotid plaques predicted adverse events (hazard ratio (HR) 2.52, 95% confidence interval (CI) 1.17-5.46; P = 0.019). In multivariable analysis, the presence of carotid plaques remained a predictor of adverse events (HR 2.26, 95%CI 1.04-4.95, P = 0.041). Other predictors identified were glomerular filtration rate, metabolic equivalents and moderate mitral regurgitation.
This study demonstrates that subclinical atherosclerosis defined by carotid plaques predicts adverse events in obese patients with negative EE.
To the best of our knowledge, this is the first article that correlates subclinical atherosclerosis and adverse events in obese patients, and specifically in obese patients with suspicion of ischaemic heart disease and a good prognosis from EE. One lesson from this study is that these good prognosis patients could be further stratified with carotid imaging and, in the case of plaque presence, benefit from more aggressive prevention measures.