Published online Dec 26, 2015. doi: 10.4330/wjc.v7.i12.875
Peer-review started: June 4, 2015
First decision: August 8, 2015
Revised: September 13, 2015
Accepted: October 23, 2015
Article in press: October 27, 2015
Published online: December 26, 2015
Processing time: 205 Days and 8.6 Hours
Prosthetic valve obstruction (PVO) is a rare but feared complication of mechanical valve replacement. Diagnostic evaluation should focus on differentiating prosthetic valve thrombosis (PVT) from pannus formation, as their treatment options differ. History of sub-optimal anti-coagulation and post-op time course to development of PVO are useful clinical characteristics in differentiating thrombus from pannus formation. Treatment of PVT is influenced by the patient’s symptoms, valve location, degree of obstruction and thrombus size and may include thrombolysis or surgical intervention. Alternatively, pannus formation requires surgical intervention. The purpose of this article is to review the pathophysiology, epidemiology, diagnostic approach and treatment options for aortic and mitral valve PVO.
Core tip: Prosthetic valve obstruction (PVO), while rare, is a dreaded complication of mechanical valve replacement. Careful clinical and multiple non-invasive imaging modalities are necessary to assess suspected PVO and evaluate for pannus overgrowth or valve thrombosis. Unlike pannus overgrowth, prosthetic valve thrombosis is more common, occurs earlier in the post-op period, is frequently related to inadequate anti-coagulation, and can often be treated through non-invasive thrombolysis. While the current understanding of pannus overgrowth remains elusive, future clarification of its pathophysiology may allow for the development of non-invasive therapeutic options.