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World J Biol Chem. Sep 26, 2010; 1(9): 286-290
Published online Sep 26, 2010. doi: 10.4331/wjbc.v1.i9.286
Benfang Lei’s research on heme acquisition in Gram-positive pathogens and bacterial pathogenesis
Benfang Lei
Benfang Lei, Department of Veterinary Molecular Biology, Montana State University, 960 Technology Blvd, Bozeman, MT 59717, United States
Author contributions: Lei B solely contributed to this manuscript.
Supported by National Institutes of Health Grants AI057347 and P20 RR-020185, US Department of Agriculture National Research Initiative/Competitive Grants Program Grants 2006-01690 and 2007-35204-18306, Montana Board of Research and Commercialization Technology Grant #07-51, USDA Animal Health Formula Funds, and the Montana State Agricultural Experiment Station
Correspondence to: Benfang Lei, PhD, Department of Veterinary Molecular Biology, Montana State University, 960 Technology Blvd, PO Box 173610, Bozeman, MT 59717, United States. blei@montana.edu
Telephone: +1-406-9946389 Fax: +1-406-9944303
Received: July 3, 2010
Revised: August 20, 2010
Accepted: August 27, 2010
Published online: September 26, 2010
Abstract

Benfang Lei’s laboratory conducts research on pathogenesis of human pathogen Group A Streptococcus (GAS) and horse pathogen Streptococcus equi (S. equi). His current research focuses on heme acquisition in Gram-positive pathogens and molecular mechanism of GAS and S. equi pathogenesis. Heme is an important source of essential iron for bacterial pathogens. Benfang Lei and colleagues identified the first cell surface heme-binding protein in Gram-positive pathogens and the heme acquisition system in GAS, demonstrated direct heme transfer from one protein to another, demonstrated an experimental pathway of heme acquisition by the Staphylococcus aureus Isd system, elucidated the activated heme transfer mechanism, and obtained evidence for a chemical mechanism of direct axial ligand displacement during the Shp-to-HtsA heme transfer reaction. These findings have considerably contributed to the progress that has been made over recent years in understanding the heme acquisition process in Gram-positive pathogens. Pathogenesis of GAS is mediated by an abundance of extracellular proteins, and pathogenic role and functional mechanism are not known for many of these virulence factors. Lei laboratory identified a secreted protein of GAS as a CovRS-regulated virulence factor that is a protective antigen and is critical for GAS spreading in the skin and systemic dissemination. These studies may lead to development of novel strategies to prevent and treat GAS infections.

Keywords: Bacterial pathogenesis; Virulence factor; Innate immunity; Group A Streptococcus; Streptococcus equi; Esterase; Heme acquisition; Heme binding protein