Published online Feb 27, 2024. doi: 10.4240/wjgs.v16.i2.463
Peer-review started: December 12, 2023
First decision: January 2, 2024
Revised: January 4, 2024
Accepted: January 30, 2024
Article in press: January 30, 2024
Published online: February 27, 2024
Processing time: 75 Days and 6.4 Hours
Colon cancer (CC) has a high incidence rate. Radical resection is the main treatment method for CC; however, liver metastasis (LM) often occurs post-surgery. The liver contains both innate and adaptive immune cells that monitor and remove abnormal cells and pathogens. Before LM, tumor cells secrete cytokines and exosomes to adjust the immune microenvironment of the liver, thus forming an inhibitory immune microenvironment for colonization by circulating tumor cells. This indicates that the immune state of patients with CC plays a crucial role in the occurrence and progression of LM.
To observe and analyze the relationship between immune status and expression of tumor factors in patients with LM of CC, and to provide a scientific interven
A retrospective analysis was performed. The baseline data of 100 patients with CC and 100 patients with CC who suffered from postoperative LM and were admitted to our hospital from May 2021 to May 2023 were included in the non-occurrence and occurrence groups, respectively. The immune status of the pa
Compared with the non-occurrence group, the expression of serum carcinoembryonic antigen (CEA), CA19-9, CA242, CA72-4 and CA50 in patients in the occurrence group were significantly higher, while the expression of CD3+, CD4+, CD8+, natural killer (NK) and CD4+/CD25 in patients in the occurrence group were significantly lower (P < 0.05). No significant difference was observed in other baseline data between groups (P > 0.05). Multivariate logistic regression model analysis revealed that the expressions of CEA, CA19-9, CA242, CA72-4, CA50, CD3+, CD4+, CD8+, NK, and CD4+/CD25 were associated with the LM in patients with CC. High expressions of serum CEA, CA19-9, CA242, CA72-4 and CA50, and low expressions of CD3+, CD4+, CD8+, NK, and CD4+/CD25 in patients with CC were risk factors for LM (OR > 1, P < 0.05). The receiver operating characteristic curve showed that the area under curve for CEA, CA19-9, CA242, CA72-4, CA50, CD3+, CD4+, CD8+, NK, and CD4+/CD25 in the prediction of LM in patients with CC were all > 0.80, with a high predictive value.
The expression of tumor factors and immune state-related indices in patients with CC is closely associated with the occurrence of LM.
Core Tip: Postoperative liver metastasis (LM) in patients with colon cancer (CC) leads to a poor prognosis; therefore, monitoring the risk factors that affect postoperative LM in patients with CC is crucial to improving their prognosis. The anti-tumor immune system of the body includes cellular immunity mediated by T cells and their subsets. An imbalance in the proportion of these cells or abnormalities in their function lead to a disordered immune system and a variety of immune-mediated diseases. Therefore, a correlation between postoperative LM and immune function may occur in patients with CC.