Published online Feb 27, 2024. doi: 10.4240/wjgs.v16.i2.289
Peer-review started: December 1, 2023
First decision: December 7, 2023
Revised: December 19, 2023
Accepted: January 15, 2024
Article in press: January 15, 2024
Published online: February 27, 2024
Processing time: 86 Days and 8.9 Hours
Phospholipase A2 (PLA2) enzymes are pivotal in various biological processes, such as lipid mediator production, membrane remodeling, bioenergetics, and maintaining the body surface barrier. Notably, these enzymes play a significant role in the development of diverse tumors.
To systematically and comprehensively explore the expression of the PLA2 family genes and their potential implications in cholangiocarcinoma (CCA).
We conducted an analysis of five CCA datasets from The Cancer Genome Atlas and the Gene Expression Omnibus. The study identified differentially expressed genes between tumor tissues and adjacent normal tissues, with a focus on PLA2G2A and PLA2G12B. Gene Set Enrichment Analysis was utilized to pinpoint associated pathways. Moreover, relevant hub genes and microRNAs for PLA2G2A and PLA2G12B were predicted, and their correlation with the pro
PLA2G2A and PLA2G12B were discerned as differentially expressed in CCA, manifesting significant variations in expression levels in urine and serum between CCA patients and healthy individuals. Elevated expression of PLA2G2A was correlated with poorer overall survival in CCA patients. Additionally, the study delineated pathways and miRNAs associated with these genes.
Our findings suggest that PLA2G2A and PLA2G12B may serve as novel potential diagnostic and prognostic markers for CCA. The increased levels of these genes in biological fluids could be employed as non-invasive markers for CCA, and their expression levels are indicative of prognosis, underscoring their potential utility in clinical settings.
Core Tip: This study reveals significant findings in cholangiocarcinoma (CCA) research, focusing on the roles of PLA2G2A and PLA2G12B enzymes. Key discoveries include the elevated expression of these enzymes in CCA, their involvement in carcinogenic pathways, and their potential as diagnostic and prognostic biomarkers. Notably, PLA2G2A’s expression correlates with poor survival outcomes in CCA patients. Additionally, the study highlights the prognostic value of associated microRNAs in serum and urine, offering new insights into non-invasive biomarkers for CCA. The research underscores the need for further validation and exploration of these findings in clinical settings.