Published online Jul 27, 2023. doi: 10.4240/wjgs.v15.i7.1304
Peer-review started: January 30, 2023
First decision: March 24, 2023
Revised: April 6, 2023
Accepted: April 21, 2023
Article in press: April 21, 2023
Published online: July 27, 2023
Processing time: 172 Days and 0.3 Hours
Different metabolic/bariatric surgery approaches vary in their effect on weight loss and glucose levels, although the underlying mechanism is unclear. Studies have demonstrated that the gut microbiota might be an important mechanism of improved metabolism after metabolic/bariatric surgery.
To investigate the relationship between the improvement in metabolic distur
We performed sleeve gastrectomy (SG), distal small intestine bypass (DSIB) or sham surgery in nonobese rats with diabetes induced by 60 mg/kg streptozotocin (STZ-DM).
The group comparisons revealed that both SG and DSIB induced a reduction in body weight and significant improvements in glucose and lipid metabolism in the STZ-DM rats. Furthermore, DSIB exhibited a stronger glucose-lowering and lipid-reducing effect on STZ-DM rats than SG. 16S ribosomal RNA gene sequencing revealed the gut abundance of some Lactobacillus spp. increased in both the SG and DSIB groups after surgery. However, the DSIB group exhibited a more pronounced increase in the gut abundance of Lactobacillus spp. compared to the SG group, with more Lactobacillus spp. types increased in the gut.
The gut abundance of Lactobacillus was significantly correlated with the improvement in glycolipid metabolism and the change in serum fibroblast growth factor 21 levels.
Core Tip: Sleeve gastrectomy and distal small intestine bypass induced a reduction in body weight and significant improvements in glucose and lipid metabolism in the rats with streptozotocin-induced nonobese diabetes. The gut abundance of some Lactobacillus spp. increased in both the sleeve gastrectomy and distal small intestine bypass groups after surgery. The gut abundance of Lactobacillus was significantly correlated with the improvement in glycolipid metabolism and the change in serum fibroblast growth factor 21 levels.