Pathophysiological role and therapeutic implications of inflammation in diabetic nephropathy

doi:10.4239/wjd.v3.i1.7

Advanced Search

BPG is committed to discovery and dissemination of knowledge

Home / Archive / Volume 3, Issue 1

This Article

Citation of this article

Corresponding Author of This Article

Article-Type of This Article

Open-Access Policy of This Article

Times Cited Counts in Google of This Article

Number of Hits and Downloads for This Article

Total Article Views (13772)

All Articles published online

The chart showing PDF series, HTML series, Figures (1-2) series, Tables (1-2) series.

Item

Count

PDF

890

HTML

11416

Figures (1-2)

1062

Tables (1-2)

404

Sum=13772

Jan 15, 2012 (publication date) through Jan 6, 2025

Times Cited of This Article

Times Cited (92)

Journal Information of This Article

Publication Name

World Journal of Diabetes

ISSN

1948-9358

Publisher of This Article

Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

Review

World J Diabetes. Jan 15, 2012; 3(1): 7-18
Published online Jan 15, 2012. doi: 10.4239/wjd.v3.i1.7

Full Size Figure Download Figure

Figure 1 Schematic overview of the participation of inflammatory mechanisms in the pathophysiology of diabetic nephropathy. MCP: Monocyte chemoattractant protein; CSF: Colony-stimulating factor; ICAM: Intercelular adhesion molecule; VCAM: Vascular cell adhesion molecule. TGF: Transforming growth factor; CTGF: Connective tissue growth factor; VEGF: Vascular endothelial growth factor; TNF-α: Tumor necrosis factor alpha.

Citation: Luis-Rodríguez D, Martínez-Castelao A, Górriz JL, Álvaro F, Navarro-González JF. Pathophysiological role and therapeutic implications of inflammation in diabetic nephropathy. World J Diabetes 2012; 3(1): 7-18
URL: https://www.wjgnet.com/1948-9358/full/v3/i1/7.htm
DOI: https://dx.doi.org/10.4239/wjd.v3.i1.7