Published online May 15, 2015. doi: 10.4239/wjd.v6.i4.554
Peer-review started: August 28, 2014
First decision: December 17, 2014
Revised: December 30, 2014
Accepted: February 10, 2015
Article in press: February 12, 2015
Published online: May 15, 2015
Processing time: 260 Days and 11.4 Hours
Diabetes affects at least 382 million people worldwide and the incidence is expected to reach 592 million by 2035. The incidence of diabetes in youth is skyrocketing as evidenced by a 21% increase in type 1 diabetes and a 30.5% increase in type 2 diabetes in the United States between 2001 and 2009. The effects of toxic stress, the culmination of biological and environmental interactions, on the development of diabetes complications is gaining attention. Stress impacts the hypothalamus-pituitary-adrenal axis and contributes to inflammation, a key biological contributor to the pathogenesis of diabetes and its associated complications. This review provides an overview of common diabetic complications such as neuropathy, cognitive decline, depression, nephropathy and cardiovascular disease. The review also provides a discussion of the role of inflammation and stress in the development and progression of chronic complications of diabetes, associated symptomatology and importance of early identification of symptoms of depression, fatigue, exercise intolerance and pain.
Core tip: The incidence of diabetes and associated complications are increasing. Toxic stress and inflammation may be contributors to the development and progression of diabetes complications. Current evidence supports early identification of symptoms of toxic stress for preventative strategies of associated risks for diabetes complications as well as assessment of the exacerbation of symptoms related to neuropathy, cardiovascular disease and nephropathy.