Published online Jun 15, 2024. doi: 10.4239/wjd.v15.i6.1086
Revised: February 1, 2024
Accepted: March 25, 2024
Published online: June 15, 2024
Processing time: 156 Days and 6.4 Hours
Diabetic nephropathy (DN) is the leading cause of end-stage renal disease and is also associated with increased risk for cardiovascular events. Until recently, strict glycemic control and blockade of the renin-angiotensin system (RAS) constituted the mainstay of treatment of DN. However, randomized controlled trials showed that sodium-glucose cotransporter 2 inhibitors further reduce the progression of DN. Therefore, these agents are recommended in all patients with DN regardless of DN stage and HbA1c levels. Moreover, additional blockade of the RAS with finerenone, a selective non-steroidal mineralocorticoid receptor antagonist, was also shown to prevent both the decline of renal function and cardiovascular events in this population. Finally, promising preliminary findings suggest that glucagon-like peptide 1 receptor agonists might also exert reno- and cardioprotective effects in patients with DN. Hopefully, this knowledge will improve the outcomes of this high-risk group of patients.
Core Tip: The management of diabetic nephropathy evolved substantially in recent years with the accumulation of evidence showing that sodium-glucose cotransporter 2 inhibitors and potentially finerenone prevent both renal function deterioration and cardiovascular events.