Published online Nov 15, 2024. doi: 10.4239/wjd.v15.i11.2203
Revised: September 3, 2024
Accepted: September 18, 2024
Published online: November 15, 2024
Processing time: 227 Days and 5.4 Hours
Gestational diabetes mellitus (GDM) women require prenatal care to minimize short- and long-term complications. The mechanism by which exercise during pregnancy affects organ development and whether glucose transporter (GLUT) 1 plays a role in GDM offspring organ development remains unknown.
To determine the effect of exercise during pregnancy on the cardiac, hepatic and renal development of GDM mother’s offspring.
Placenta samples were collected from humans and mice. GDM mouse models were created using streptozotocin along with a GDM with exercise group. The hearts, livers and kidneys of 3- and 8-week-old offspring were collected for body composition analysis and staining. The effects of high glucose levels and hypoxia were investigated using HTR8/SVneo. Transwell and wound-healing assays were performed to assess cell migration. Immunofluorescence accompanied with TUNEL and Ki67 staining was used to explore apoptosis and proliferation.
Exercise during pregnancy downregulated the GLUT1 and hypoxia inducible factor-1α expression in placenta from individuals with GDM. Cobalt chloride-induced hypoxia and high glucose levels also significantly decreased migration and apoptosis of HTR8/SVneo cells. In addition, exercise reduced inflammatory cell infiltration in the liver and decreased the tubular vacuolar area in the kidneys of offspring.
GDM affects the growth and development of organs in offspring. Exercise during pregnancy can reverse adverse effects of GDM on the development of the heart, liver, and kidney in offspring.
Core Tip: We established a mouse model of gestational diabetes mellitus (GDM) and an exercise model during pregnancy. We discussed placental glucose transporter (GLUT) 1 expression and the hypoxic microenvironment, and the correlation between them. We also investigated organ development in the heart, liver, and kidney of the offspring. Our findings suggest that exercise during pregnancy is beneficial for organ development in the offspring of GDM. This benefit may be realized by modulating GLUT 1 expression and improving placental hypoxia. We hope that this study will emphasize the importance of exercise during pregnancy in women with GDM to improve its effects on organ development in the offspring.