Published online Nov 15, 2024. doi: 10.4239/wjd.v15.i11.2157
Revised: August 29, 2024
Accepted: September 26, 2024
Published online: November 15, 2024
Processing time: 195 Days and 21 Hours
In this editorial, we comment on an article by Tang et al published in the World Journal of Diabetes. Obesity and diabetes are two pathological situations that are intrinsically related. Neither lifestyle changes nor pharmacological treatments have achieved diabetes remission. From this perspective, bariatric surgery has been widely used as an approach for weight loss in obese patients and as a strategy to promote metabolic modulation. The main effects of bariatric surgery involve direct action in improving cardiovascular function and endothelial function and reducing insulin resistance, leading to diabetes remission in the short term following surgery. In this context, it has been observed that hormones from the gastrointestinal tract and endothelium play a prominent role in this process. By reversing endothelial dysfunction, it is possible to balance pro-inflammatory cytokine production, improving the availability of nitric oxide and inhibiting vascular oxidative stress. Furthermore, it can be considered an efficient anti-inflammatory strategy, alleviating interferon-gamma-mediated adipose tissue inflammation. The current challenge must be to unravel the pathophysiological mechanisms and potential targets for treating metabolic diseases.
Core Tip: In this editorial, we address the pathophysiological mechanisms associated with bariatric surgery in type 2 diabetes remission. Within this perspective, we discuss gastrointestinal tract hormones, mainly peptide-1, which is a hormone secreted by gastrointestinal L cells and released immediately after food intake, as well as the benefits obtained after reversing endothelial syndrome via bariatric surgery.