Published online Nov 15, 2021. doi: 10.4239/wjd.v12.i11.1856
Peer-review started: May 5, 2021
First decision: July 15, 2021
Revised: July 29, 2021
Accepted: October 9, 2021
Article in press: October 9, 2021
Published online: November 15, 2021
Processing time: 193 Days and 20.5 Hours
Cardiovascular disease is the predominant cause of death in type 2 diabetes mellitus (T2DM). Evidence suggests a strong association between duration and degree of hyperglycemia and vascular disease. However, large trials failed to show cardiovascular benefit after intensive glycemic control, especially in patients with longer diabetes duration. Atherosclerosis is a chronic and progressive disease, with a long asymptomatic phase. Subclinical atherosclerosis, which is impaired in T2DM, includes impaired vasodilation, increased coronary artery calcification (CAC), carotid intima media thickness, arterial stiffness, and reduced arterial elasticity. Each of these alterations is represented by a marker of subclinical atherosclerosis, offering a cost-effective alternative compared to classic cardiac imaging. Their additional use on top of traditional risk assessment strengthens the predictive risk for developing coronary artery disease (CAD). We, herein, review the existing literature on the effect of glycemic control on each of these markers separately. Effective glycemic control, especially in earlier stages of the disease, attenuates progression of structural markers like intima-media thickness and CAC. Functional markers are improved after use of newer anti-diabetic agents, such as incretin-based treatments or sodium-glucose co-transporter-2 inhibitors, especially in T2DM patients with shorter disease duration. Larger prospective trials are needed to enhance causal inferences of glycemic control on clinical endpoints of CAD.
Core Tip: Progression or even regression of atherosclerosis is possible in type 2 diabetes mellitus, especially at an early stage of the disease, with better glycemic control and use of newer agents, such as dipeptidyl peptidase 4 inhibitors and sodium-glucose co-transporter-2 inhibitors. Despite considerable evidence, especially for structural markers like intima media thickness or coronary artery calcification, and pulse wave velocity, larger and longer trials are needed to establish their clinical utility and correlation with clinical end-points.