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Cited by in CrossRef
For: Caputo V, Termine A, Strafella C, Giardina E, Cascella R. Shared (epi)genomic background connecting neurodegenerative diseases and type 2 diabetes. World J Diabetes 2020; 11(5): 155-164 [PMID: 32477452 DOI: 10.4239/wjd.v11.i5.155]
URL: https://www.wjgnet.com/1948-9358/full/v11/i5/155.htm
Number Citing Articles
1
Dongmei Wu, Yang Hu, Min Song, Gongbo Li. Dichlorodiphenyltrichloroethane Impairs Amyloid Beta Clearance by Decreasing Liver X Receptor α ExpressionFrontiers in Aging Neuroscience 2021; 13 doi: 10.3389/fnagi.2021.634948
2
Arwa M. Amin, Hamza Mostafa, Hani M.J. Khojah. Insulin resistance in Alzheimer’s disease: The genetics and metabolomics linksClinica Chimica Acta 2023; 539: 215 doi: 10.1016/j.cca.2022.12.016
3
Qianxu Ren, Junling Fu, Xiaoye Duan, Lina Sun, Zhijing Mu, Wenping Liang, Yu Li, Zhe Wang, Shuangling Xiu. The Effects of Ketogenic Diet on Brain Gene Expressions in Type 2 Diabetes BackgroundNeuroscience 2024; 549: 101 doi: 10.1016/j.neuroscience.2024.04.008
4
Wided Boukhalfa, Haifa Jmel, Nadia Kheriji, Ismail Gouiza, Hamza Dallali, Mariem Hechmi, Rym Kefi. Decoding the genetic relationship between Alzheimer’s disease and type 2 diabetes: potential risk variants and future direction for North AfricaFrontiers in Aging Neuroscience 2023; 15 doi: 10.3389/fnagi.2023.1114810
5
Zahra Salehi, Masoud Arabfard, Omid Sadatpour, Mina Ohadi. Proposed minimal essential co-expression and physical interaction networks involved in the development of cognition impairment in human mid and late lifeNeurological Sciences 2021; 42(3): 951 doi: 10.1007/s10072-020-04594-0
6
Xinzhu Yu, Artitaya Lophatananon, Krisztina Mekli, Alistair Burns, Kenneth R. Muir, Hui Guo. A suggested shared aetiology of dementia - a colocalization studyNeurobiology of Aging 2022; 117: 71 doi: 10.1016/j.neurobiolaging.2022.05.005
7
Jiaqi Yang, Yuanfeng Huang, Zheng Wang, Shiyu Zhang, Dai Wu, Jiayi Xiong, Heng Wu, Yijing Wang, Qiao Zhou, Yixiao Zhu, Guihu Zhao, Bin Li, Jifeng Guo, Kun Xia, Beisha Tang, Jinchen Li. A PheWAS approach to identify associations of GBA1 variants with comprehensive phenotypes beyond neurological diseasesnpj Parkinson's Disease 2025; 11(1) doi: 10.1038/s41531-025-00901-8