Copyright
©The Author(s) 2021.
World J Gastrointest Oncol. Aug 15, 2021; 13(8): 835-844
Published online Aug 15, 2021. doi: 10.4251/wjgo.v13.i8.835
Published online Aug 15, 2021. doi: 10.4251/wjgo.v13.i8.835
Figure 1 The possible roles of bacteria, including Helicobacter pylori, and other factors in pancreatic cancer development.
Figure 2 The potential mechanism by which Helicobacter pylori might contribute to autoimmune pancreatitis.
H. pylori: Helicobacter pylori; GP: Glycoproteins; Hsp: Heat shock protein; H+/K+ ATPase: H+/K+-adenosine triphosphatase; HLA-DR: Human leukocyte class II DR antigens; CA-II: Carbonic anhydrase type II antigens; ACA-II: Anticarbonic anhydrase II antibody; LF: Lactoferrin; ALF: Antilactoferrin antibody; TLs: T lymphocytes; IFN-ɣ: Interferon-ɣ; anti-Hsps: Antibodies against heat shock proteins; ROMs: Reactive oxygen metabolites.
- Citation: Kunovsky L, Dite P, Jabandziev P, Dolina J, Vaculova J, Blaho M, Bojkova M, Dvorackova J, Uvirova M, Kala Z, Trna J. Helicobacter pylori infection and other bacteria in pancreatic cancer and autoimmune pancreatitis. World J Gastrointest Oncol 2021; 13(8): 835-844
- URL: https://www.wjgnet.com/1948-5204/full/v13/i8/835.htm
- DOI: https://dx.doi.org/10.4251/wjgo.v13.i8.835