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©The Author(s) 2018.
World J Gastrointest Oncol. Jun 15, 2018; 10(6): 124-136
Published online Jun 15, 2018. doi: 10.4251/wjgo.v10.i6.124
Published online Jun 15, 2018. doi: 10.4251/wjgo.v10.i6.124
Figure 1 Schema of molecules associated with each step of the establishment of hepatic metastasis from gastric cancer.
VIM: Vimentin; GPR155: G protein-coupled receptor 155; HIF-1α: Hypoxia inducible factor-1 alpha; EGFL7: Epidermal growth factor-like domain-containing protein 7; CXCL1: C-X-C motif chemokine ligand 1; TIMP1: Tissue inhibitor of metallopeptidase 1; NFKB1/p105: Nuclear factor kappa B subunit 1; MAP1LC3: Microtubule associated protein 1 light chain 3; BECN1: Beclin1; SQSTM1/p62: Sequestosome 1; MFSD4: Major facilitator superfamily domain containing 4; PAK1: P21 (RAC1) activated kinase 1; VEGF-D: Vascular endothelial growth factor-D; TYMP: Thymidine phosphorylase.
- Citation: Shimizu D, Kanda M, Kodera Y. Emerging evidence of the molecular landscape specific for hematogenous metastasis from gastric cancer. World J Gastrointest Oncol 2018; 10(6): 124-136
- URL: https://www.wjgnet.com/1948-5204/full/v10/i6/124.htm
- DOI: https://dx.doi.org/10.4251/wjgo.v10.i6.124