Published online Jan 27, 2024. doi: 10.4254/wjh.v16.i1.41
Peer-review started: November 21, 2023
First decision: December 5, 2023
Revised: December 18, 2023
Accepted: January 9, 2024
Article in press: January 9, 2024
Published online: January 27, 2024
Processing time: 63 Days and 0.2 Hours
Direct-acting antivirals (DAAs) revolutionized the treatment of chronic hepatitis C virus (HCV)-associated disease achieving high rates of sustained virological response (SVR). However, whether DAAs can reduce the occurrence of hepatocellular carcinoma (HCC) in patients with HCV-associated cirrhosis who are at high risk have not been concluded.
The key to the retrospective cohort study is to explore DAA treatment in HCV-associated cirrhosis patients with HCC. Solutions to optimize DAAs treatment are explored to reduce the occurrence of HCC in patients with HCV-associated cirrhosis, and careful follow-up is needed.
To investigate the effect of DAAs on the occurrence of HCC in patients with HCV-associated cirrhosis after achieving SVR.
427 inpatients with HCV-associated cirrhosis were enrolled in Tianjin Second People's Hospital from January 2014 to April 2020. 118 patients weren’t received antiviral treatment with any reasons named non-antiviral treatment group, and 236 patients obtained from the 309 DAAs treatment patients according to the propensity score matching named DAAs treatment group. Demographic information and laboratory data were collected from baseline and the following up. Kaplan-Meier curve and Log-Rank test were used to compare the incidence and cumulative incidence of HCC between the two groups. Cox proportional risk regression was used to re-evaluate the risk factors for HCC.
The DAA treatment group was followed up for 1-84 mo, with a median follow-up of 28 mo, while the non-antiviral treatment group was followed up for 5-84 mo, with a median follow-up of 37 mo. Age > 58 [hazard ratio (HR) = 1.089; 95% confidence interval (CI), 1.033-1.147; P = 0.002] and liver stiffness measurement > 27.85 kPa (HR = 1.043; 95%CI, 1.022-1.065; P = 0.000) were risk factors for HCC in all patients (n = 427), and DAA treatment didn’t show protective efficacy. After adjusting for confounding factors (age and sex), 27 cases of HCC occurred in the new DAA treatment group (236 cases), and there was no significant difference in the total incidence of HCC between the two groups (χ2 = 0.369, P = 0.544). In the new DAA treatment group, HCC incidence was 4.68/100PY (95%CI, 3.09-6.81), while it was 3.00/100PY (95%CI, 1.50-5.37) in the non-antiviral treatment group. The follow-up time of the new DAA treatment group was 1-84 mo (29.33 ± 16.20), the median follow-up time was 27 mo and the time of HCC occurrence in the new DAA treatment group was 5-66 mo. The incidence of HCC at 12, 24, 36 and 48 mo was 3.39%, 6.36%, 8.47% and 10.17% in the new DAA treatment group, and it was 0%, 0%, 3.39% and 9.32% in the non-antiviral treatment group, respectively.
This is a novel assessment that provides theoretical insight into the impact of achieving SVR after DAA on HCC development in patients with HCV-associated cirrhosis. This study found that DAAs did not reduce the incidence of HCC in HCV-associated cirrhosis compared with no antiviral therapy, suggesting that the clinical priority of DAAs for patients with HCV is justified. We should also explore solutions to optimize DAAs therapy to reduce the occurrence of HCC in patients with HCV-associated cirrhosis.
In future study, we should be focused on the research of the multicenter, large data, in order to more accurately assess DAAs influence on HCV-associated liver diseases in patients with HCC, thereby reducing the occurrence of HCC, and it can from common biochemical indicator, liquid biopsy, multiple sets of multi-angle discussion such as HCV-associated liver disease risk factors in patients with HCC.