Published online Jan 27, 2015. doi: 10.4254/wjh.v7.i1.121
Peer-review started: July 17, 2014
First decision: July 29, 2014
Revised: August 15, 2014
Accepted: November 7, 2014
Article in press: November 10, 2014
Published online: January 27, 2015
Processing time: 177 Days and 21.2 Hours
The consequences of hepatitis B virus (HBV) and human immunodeficiency virus (HIV) co-infection on progression of severe liver diseases is a serious public health issue, worldwide. In the co-infection cases, about 90% of HIV-infected population is seropositive for HBV where approximately 5%-40% individuals are chronically infected. In HIV co-infected individuals, liver-related mortality is estimated over 17 times higher than those with HBV mono-infection. The spectrum of HIV-induced liver diseases includes hepatitis, steatohepatitis, endothelialitis, necrosis, granulomatosis, cirrhosis and carcinoma. Moreover, HIV co-infection significantly alters the natural history of hepatitis B, and therefore complicates the disease management. Though several studies have demonstrated impact of HIV proteins on hepatocyte biology, only a few data is available on interactions between HBV and HIV proteins. Thus, the clinical spectrum as well as the complexity of the co-infection offers challenging fronts to study the underlying molecular mechanisms, and to design effective therapeutic strategies.
Core tip: The consequences of hepatitis B virus (HBV) and human immunodeficiency virus (HIV) co-infection on progression of severe liver diseases is a serious public health issue, worldwide. In HIV co-infected individuals, liver-related mortality is estimated over 17 times higher than those with HBV mono-infection. HIV co-infection significantly alters the natural history of hepatitis B, and therefore complicates the disease management. Thus, the clinical spectrum as well as the complexity of the HBV and HIV co-infection of liver offers challenging fronts to study the underlying molecular mechanisms, and to design effective therapeutic strategies.