Review
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World J Hepatol. Jun 27, 2014; 6(6): 370-383
Published online Jun 27, 2014. doi: 10.4254/wjh.v6.i6.370
Management of cytomegalovirus infection and disease in liver transplant recipients
Jackrapong Bruminhent, Raymund R Razonable
Jackrapong Bruminhent, Division of Infectious Diseases, College of Medicine, Mayo Clinic, Rochester, MN 55905, United States
Raymund R Razonable, Division of Infectious Diseases and the William J von Liebig Center for Transplantation and Clinical Regeneration, College of Medicine, Mayo Clinic, Rochester, MN 55905, United States
Author contributions: Bruminhent J and Razonable RR contributed equally to this review paper.
Correspondence to: Raymund R Razonable, MD, Division of Infectious Diseases and the William J von Liebig Center for Transplantation and Clinical Regeneration, College of Medicine, Mayo Clinic, Marian Hall 5th Floor, 200 First Street SW, Rochester, MN 55905, United States. razonable.raymund@mayo.edu
Telephone: +1-507-2843747 Fax: +1-507-2557767
Received: November 12, 2013
Revised: January 23, 2014
Accepted: March 13, 2014
Published online: June 27, 2014
Processing time: 234 Days and 16.9 Hours
Abstract

Cytomegalovirus (CMV) is one of the most common viral pathogens causing clinical disease in liver transplant recipients, and contributing to substantial morbidity and occasional mortality. CMV causes febrile illness often accompanied by bone marrow suppression, and in some cases, invades tissues including the transplanted liver allograft. In addition, CMV has been significantly associated with an increased predisposition to acute and chronic allograft rejection, accelerated hepatitis C recurrence, and other opportunistic infections, as well as reduced overall patient and allograft survival. To negate the adverse effects of CMV infection on transplant outcome, its prevention, whether through antiviral prophylaxis or preemptive therapy, is an essential component to the management of liver transplant recipients. Two recently updated guidelines have suggested that antiviral prophylaxis or preemptive therapy are similarly effective in preventing CMV disease in modest-risk CMV-seropositive liver transplant recipients, while antiviral prophylaxis is the preferred strategy over preemptive therapy for the prevention of CMV disease in high-risk recipients [CMV-seronegative recipients of liver allografts from CMV-seropositive donors (D+/R-)]. However, antiviral prophylaxis has only delayed the onset of CMV disease in many CMV D+/R- liver transplant recipients, and such occurrence of late-onset CMV disease was significantly associated with increased all-cause and infection-related mortality after liver transplantation. Therefore, a search for better strategies for prevention, such as prolonged duration of antiviral prophylaxis, a hybrid approach (antiviral prophylaxis followed by preemptive therapy), or the use of immunologic measures to guide antiviral prophylaxis has been suggested to prevent late-onset CMV disease. The standard treatment of CMV disease consists of intravenous ganciclovir or oral valganciclovir, and if feasible, reduction in pharmacologic immunosuppression. In one clinical trial, oral valganciclovir was as effective as intravenous ganciclovir for the treatment of mild to moderate CMV disease in solid organ (including liver) transplant recipients. The aim of this article is to provide a state-of-the art review of the epidemiology, diagnosis, prevention, and treatment of CMV infection and disease after liver transplantation.

Keywords: Cytomegalovirus; Outcome; Hepatitis; Transplantation; Valganciclovir; Prophylaxis; Treatment

Core tip: This paper summarizes the current state in the management of cytomegalovirus disease after liver transplantation, including a review of recently updated guidelines for diagnosis, prevention and treatment.