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Dec 27, 2014 (publication date) through Nov 27, 2024
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World Journal of Hepatology
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1948-5182
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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA
| For: | Takeuchi M, Takino JI, Sakasai-Sakai A, Takata T, Ueda T, Tsutsumi M, Hyogo H, Yamagishi SI. Involvement of the TAGE-RAGE system in non-alcoholic steatohepatitis: Novel treatment strategies. World J Hepatol 2014; 6(12): 880-893 [PMID: 25544875 DOI: 10.4254/wjh.v6.i12.880] |
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| URL: | https://www.wjgnet.com/1948-5182/full/v6/i12/880.htm |
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Masayoshi Takeuchi. Serum Levels of Toxic AGEs (TAGE) May Be a Promising Novel Biomarker for the Onset/Progression of Lifestyle-Related Diseases. Diagnostics 2016; 6(2): 23 doi: 10.3390/diagnostics6020023
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Xin Sun, Yan Zhang, Meilin Xie. The role of peroxisome proliferator-activated receptor in the treatment of non-alcoholic fatty liver disease. Acta Pharmaceutica 2017; 67(1): 1 doi: 10.1515/acph-2017-0007
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Rohini Mehta, Gladys Shaw, Peter Masschelin, Sean Felix, Munkzhul Otgonsuren, Ancha Baranova, Zachary Goodman, Zobair Younossi, Barry I. Hudson. Polymorphisms in the receptor for advanced glycation end-products (RAGE) gene and circulating RAGE levels as a susceptibility factor for non-alcoholic steatohepatitis (NASH). PLOS ONE 2018; 13(6): e0199294 doi: 10.1371/journal.pone.0199294
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Kathleen Priken, Gladys Tapia, Cynthia Cadagan, Nicolás Quezada, Javiera Torres, Amanda D'Espessailles, Paulina Pettinelli. Higher hepatic advanced glycation end products and liver damage markers are associated with nonalcoholic steatohepatitis. Nutrition Research 2022; 104: 71 doi: 10.1016/j.nutres.2022.04.005
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Mona M. Allam, Walaa B. El gazzar. Exendin-4, a glucagon-like peptide-1 receptor agonist downregulates hepatic receptor for advanced glycation end products in non-alcoholic steatohepatitis rat model. Archives of Physiology and Biochemistry 2018; 124(1): 10 doi: 10.1080/13813455.2017.1348362
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Akiko Sakasai-Sakai, Kenji Takeda, Masayoshi Takeuchi. Involvement of Intracellular TAGE and the TAGE–RAGE–ROS Axis in the Onset and Progression of NAFLD/NASH. Antioxidants 2023; 12(3): 748 doi: 10.3390/antiox12030748
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Masayoshi Takeuchi, Jun-ichi Takino, Akiko Sakasai-Sakai, Takanobu Takata, Mikihiro Tsutsumi. Toxic AGE (TAGE) Theory for the Pathophysiology of the Onset/Progression of NAFLD and ALD. Nutrients 2017; 9(6): 634 doi: 10.3390/nu9060634
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Mona M. Allam, Reham M. Ibrahim, Walaa Bayoumie El Gazzar, Mona A. Said. Dipeptedyl peptidase-4 (DPP-4) inhibitor downregulates HMGB1/TLR4/NF-κB signaling pathway in a diabetic rat model of non-alcoholic fatty liver disease. Archives of Physiology and Biochemistry 2024; 130(1): 87 doi: 10.1080/13813455.2021.1975758
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