Mechanisms involved in Helicobacter pylori induced duodenal ulcer disease: an overview

Advanced Search

BPG is committed to discovery and dissemination of knowledge

Home / Archive / Volume 6, Issue 5

This Article

Citation of this article

Corresponding Author of This Article

Article-Type of This Article

Open-Access Policy of This Article

Times Cited Counts in Google of This Article

Number of Hits and Downloads for This Article

All Articles published online

Item

Count

PDF

310

HTML

3072

Figures (1-2)

204

Tables (1-1)

124

Sum=3710

Oct 15, 2000 (publication date) through May 4, 2024

Times Cited of This Article

Journal Information of This Article

Publication Name

World Journal of Gastroenterology

ISSN

1007-9327

Publisher of This Article

Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

Editorial

World J Gastroenterol. Oct 15, 2000; 6(5): 619-623
Published online Oct 15, 2000. doi: 10.3748/wjg.v6.i5.619

Table 1 Gastric acid secretory characteristics of duodenal patients (all of which might be Helicobacter pylori induced)

Increased maximal acid secretory capacity

Increased parietal cell mass (trophic effect of gastrin?)

Increased basal acid secretion

Increased basal release of gastrin

Increased and prolonged acid response to meals

Increased release of gastrin

Defective inhibitory mechanisms

Antral acidification

Antral distension

Fat in the duodenum (H. pylori induced?)

Citation: Olbe L, Fändriks L, Hamlet A, Svennerholm AM, Thoreson AC. Mechanisms involved in Helicobacter pylori induced duodenal ulcer disease: an overview. World J Gastroenterol 2000; 6(5): 619-623
URL: https://www.wjgnet.com/1007-9327/full/v6/i5/619.htm
DOI: https://dx.doi.org/10.3748/wjg.v6.i5.619