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©2013 Baishideng Publishing Group Co.
World J Gastroenterol. Apr 21, 2013; 19(15): 2419-2424
Published online Apr 21, 2013. doi: 10.3748/wjg.v19.i15.2419
Published online Apr 21, 2013. doi: 10.3748/wjg.v19.i15.2419
Table 1 Intraluminal pH and bile positive in the forestomach between the time of esophagoduodenostomy and 40 wk after esophagoduodenostomy
Model (n = 25) | Control (n = 29) | |||
pH (mean ± SD) | Bile positive (n) | pH (mean ± SD) | Bile positive (n) | |
At the time of esophagoduodenostomy | 3.22 ± 0.29 | 2 | 3.23 ± 0.29 | 3 |
40 wk after esophagoduodenostomy | 3.24 ± 0.31 | 2 | 3.25 ± 0.25 | 4 |
Table 2 Incidence of inflammation, intestinal metaplasia, intestinal metaplasia with dysplasia, and adenocarcinoma in the esophagus and forestomach 40 wk after surgery n (%)
Inflammation | Intestinal metaplasia | Intestinal metaplasia with dysplasia | Adenocarcinoma | |
Control esophagus (n = 29) | 2 (6.8) | 1 (3.4) | 0 | 0 |
Model esophagus (n = 25) | 24 (96) | 22 (88) | 5 (20) | 2 (8) |
Control forestomach (n = 29) | 2 (6.8) | 1 (3.4) | 0 | 0 |
Model forestomach (n = 25) | 25 (100) | 2 (8) | 0 | 0 |
- Citation: Cheng P, Li JS, Zhang LF, Chen YZ, Gong J. Exposure to gastric juice may not cause adenocarcinogenesis of the esophagus. World J Gastroenterol 2013; 19(15): 2419-2424
- URL: https://www.wjgnet.com/1007-9327/full/v19/i15/2419.htm
- DOI: https://dx.doi.org/10.3748/wjg.v19.i15.2419