Etiology and functional status of liver cirrhosis by 31P MR spectroscopy

Table 1 Concentrations of ³¹P visible metabolites (mmol/L) in the liver according to the degree of liver injury

	n	PME	Pi	PDE	ATP
Controls	11	3.09±1.45	1.63±0.55	10.83±2.68	3.72±0.99
All patients	80	3.53±1.45	1.33±0.61	7.16±2.88d	2.95±0.84b
CPS-A	18	3.64±1.68	1.37±0.56	9.16±2.32	3.24±0.85
CPS-B	25	3.60±1.31	1.31±0.57	7.31±2.62d	2.93±0.78a
CPS-C	37	3.44±1.46	1.33±0.66	6.07±2.80df	2.83±0.87b

^aP<0.05,

^bP<0.01,

^dP<0.001 vs the control group;

^fP<0.01 vs the CPS-A group.

Table 2 Concentrations (mmol/L) of ³¹P visible metabolites in the liver according to different etiologies and CPS

	n	PME	Pi	PDE	ATP	CPS	n	PME	Pi
Controls	11	3.09±1.45	1.63±0.55	10.83±2.68	3.72±0.99	-	-	-	-
Alcohol	33	3.48±1.53	1.19±0.39ac	6.52±2.29df	2.86±0.80b	A	5		3.87±2.05
						B	6	3.50±1.32	1.17±0.35
						C	22	3.38±1.51	1.21±0.43a
Viral	22	3.64±1.55	1.57±0.77c	6.47±3.13df	2.84±0.92b	A	7	3.47±1.92	1.54±0.69
						B	8	3.99±1.51	1.51±0.63
						C	7	3.40±1.41	1.66±1.04
1Cholestatic	16	3.59±1.31	1.43±0.63	9.36±2.70	3.27±0.90	A	2	-	-
						B	10	3.44±1.26	1.26±0.66
						C	4	-	-

^aP<0.05, ^bP<0.01,

^dP<0.001 vs the control group;

^cP<0.05 between alcohol and viral etiological groups;

^fP<0.001 vs the cholestatic group. 1The cholestatic group was not divided into subgroups reflecting CPS because of the insufficient number in groups CPS-A and CPS-C.