Involvement of apoptosis of alveolar epithelial cells in acute pancreatitis-associated lung injury

Figure 1 AI of alveolar epithelial cells in sodium taurocholate-induced pancreatitis-associated lung injury. AHNP was induced by retrograde injections into the pancreatic ducts of 5% sodium taurocholate (0.1 mL/100 g body wt) in rats. Results shown are the MEAN ± SEM for four or more animals in each group. Asterisks indicates ^bP < 0.01 when the AI of alveolar epithelial cells at 5 h after induction of AHNP compared with 2 and 14 h after induction of AHNP.

Figure 2 The positive rate of bax and p53 protein in alveolar epithelial cells after sodium taurocholate-induced pancreatitis-associated lung injury. AHNP was induced by retrograde injections into the pancreatic ducts of 5% sodium taurocholate (0.1 mL/100 g body wt) in rats. Results shown were the MEAN ± SEM for four or more animals in each group. Asterisks indicates ^aP < 0.05 when the positive rate of bax protein in alveolar epithelial cells at 5 h after induction of AHNP compared with 2 and 14 h and the positive rate of p53 protein in alveolar epithelial cells at 2 h after induction of AHNP compared with 5 and 14 h.

Figure 3 The positive rate of TGFβ₁ in alveolar epithelial cells after sodium taurocholate-induced pancreatitis-associated lung injury. AHNP was induced by retrograde injections into the pancreatic ducts of 5% sodium taurocholate (0.1 mL/100 g body wt) in rats. Results shown were the MEAN ± SEM for four or more animals in each group. Therewas no significant difference among the positive rate of TGFβ₁ at 2, 5 and 14 h after induction of AHNP.