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©2006 Baishideng Publishing Group Co.
World J Gastroenterol. Jan 28, 2006; 12(4): 574-581
Published online Jan 28, 2006. doi: 10.3748/wjg.v12.i4.574
Published online Jan 28, 2006. doi: 10.3748/wjg.v12.i4.574
Figure 1 Histological study of gastric mucosa in sham operative rats and rats with ischemic brain.
A: In sham operative rat stomachs irrigated with gastric juice, gastric mucosal cells appear intact; B: Disruption of the upper mucosal cells and lamina propria (indicated by arrows) in rats with ischemic brain, the injured cells are characterized by karyorrhexis and dense homogenous acidophilic cytoplasm (HE× 150)
Figure 2 Relationship between gastric mucosal ulceration and mucosal GSH levels as well as between gastric mucosal ulceration and lipid peroxide generation in gastric juice-irrigated stomachs of rats with ischemic brain.
Rat stomachs were irrigated for 3 h with simulated gastric juice.
Figure 3 Effects of L-NAME and AMG on various gastric parameters in gastric juice-irrigated stomachs of rats with ischemic brain.
Data are expressed as mean±SE (n = 8). aP < 0.05 vs vehicle.
- Citation: Hung CR. Role of gastric oxidative stress and nitric oxide in formation of hemorrhagic erosion in rats with ischemic brain. World J Gastroenterol 2006; 12(4): 574-581
- URL: https://www.wjgnet.com/1007-9327/full/v12/i4/574.htm
- DOI: https://dx.doi.org/10.3748/wjg.v12.i4.574