Published online Aug 7, 2023. doi: 10.3748/wjg.v29.i29.4557
Peer-review started: May 18, 2023
First decision: June 20, 2023
Revised: June 29, 2023
Accepted: July 19, 2023
Article in press: July 19, 2023
Published online: August 7, 2023
Processing time: 75 Days and 23.9 Hours
Recent studies have revealed that hydrogen sulfide is the third class of gas signaling molecules after nitric oxide (NO) and carbon monoxide (CO). The high level of endogenous hydrogen sulfide found in the brain, which is mainly produced by cystathionine beta-synthase, suggests that it may have a physiological function, and the nucleus tractus solitarius is important nucleus that regulates the function of internal organs, so we want to elucidate the role of hydrogen sulfide in the NTS in regulating gastric function in rats.
To investigate whether hydrogen sulfide in the nucleus tractus solitarius is involved in the regulation of gastric dysfunction by restraint water-immersion stress, this study will examine the role of hydrogen sulfide in the nucleus tractus solitarius in the regulation of gastric motility.
It is the first time to propose that hydrogen sulfide in the nucleus tractus solitarius has a regulatory effect on the gastric motility caused by restraint water-immersion stress, and to investigate its mechanism of action, which can not only elucidate the mechanism of regulation of gastric dysfunction by hydrogen sulfide in the nucleus tractus solitarius, and also provide an important experimental basis for the prevention and treatment of stress gastric ulcer from the central aspect in clinical practice.
We used immunohistochemical, fluorescent double-labeling technique and restraint water-immersion stress model to confirm the involvement of hydrogen sulfide-producing cystathionine beta-synthase neurons in the nucleus tractus solitarius in the regulation of gastric function, and physiological methods to record changes in gastric motility before and after their brain injection.
After restraint water-immersion stress, cystathionine beta-synthase neurons containing c-Fos were significantly increased and gastric motility was inhibited in rats after nucleus tractus solitarius injection of NaHS, and this inhibitory effect was eliminated after pre-injection of transient receptor potential vanilloid 1 channels, NF-κB channel blockers, and NK1 receptor antagonists followed by NaHS injection.
Injection NaHS into the nucleus tractus solitarius can inhibit gastric motility in rats and this effect may be mediated by TRPV1 and NK1 receptors via NF-κB channel-dependent activation.
Our next step will be to continue our work on the effects of endogenous hydrogen sulfide in the nucleus tractus solitarius on gastric function.