Published online Apr 28, 2023. doi: 10.3748/wjg.v29.i16.2502
Peer-review started: January 9, 2023
First decision: February 15, 2023
Revised: February 21, 2023
Accepted: March 31, 2023
Article in press: March 31, 2023
Published online: April 28, 2023
Processing time: 105 Days and 3.3 Hours
Acute cholangitis is potentially lethal when accompanied by sepsis because of biliary obstruction. It is necessary to identify predictive biomarkers for patients who require emergent biliary drainage and patients who maybe progress to systemic bloodstream infection at an early stage of the disease.
Bacteremia induces hyperactive immune response and mitochondrial dysfunction. Presepsin is responsible for pathogen recognition by innate immunity. Acylcarnitines are established mitochondrial biomarkers. However, it is unknown whether presepsin or specific acylcarnitine species can reflect the severity of acute cholangitis and the timing of biliary drainage.
To clarify the early predictive value of presepsin and acylcarnitines for severity and biliary drainage of acute cholangitis.
In this prospective observational study, 280 patients with acute cholangitis were included from May 2019 to July 2021. The severity was stratified as mild, moderate, and severe according to according to the Tokyo Guidelines 2018. Blood presepsin and plasma acylcarnitines were tested at enrollment by chemiluminescent enzyme immunoassay and ultra-high-performance liquid chromatography-mass spec
The concentrations of presepsin, procalcitonin, short- and medium-chain acylcarnitines increased, while long-chain acylcarnitines decreased with the severity of acute cholangitis. The areas under the receiver operating characteristic curves (AUC) of presepsin for diagnosing moderate/severe and severe cholangitis (0.823 and 0.801, respectively) were greater than those of conventional markers. The AUC of a combination of presepsin, direct bilirubin, alanine aminotransferase, temperature, and butyryl-L-carnitine for predicting biliary drainage was 0.723. Presepsin, procalcitonin, acetyl-L-carnitine, hydroxydodecenoyl-L-carnitine, and temperature were independent predictors of bloodstream infection. After adjusting for severity classification, acetyl-L-carnitine was the only acylcarnitine independently associated with 28-d mortality (hazard ratio 14.396; P < 0.001) (AUC: 0.880). Presepsin concentration showed positive correlation with direct bilirubin and acetyl-L-carnitine.
Presepsin may serve as a specific biomarker to predict the severity and biliary drainage of acute cholangitis. Acetyl-L-carnitine might be a promising prognostic factor for patients with acute cholangitis. Innate immune response was associated with mitochondrial metabolic dysfunction in acute cholangitis.
Prospective observational study reports the predictive value of presepsin and acylcarnitines for severity and biliary drainage of acute cholangitis. Future research should focus on the association between acylcarnitines and the changes of intestinal microflora and bacterial translocation in acute cholangitis.