Published online Oct 21, 2022. doi: 10.3748/wjg.v28.i39.5735
Peer-review started: June 22, 2022
First decision: August 1, 2022
Revised: August 12, 2022
Accepted: September 21, 2022
Article in press: September 21, 2022
Published online: October 21, 2022
Processing time: 117 Days and 23.5 Hours
Alongside respiratory symptoms, elevated liver enzymes, abnormal liver function, and even acute liver failure were reported in patients suffering from severe acute respiratory disease coronavirus 2 (SARS-CoV-2) pneumonia. However, the exact triggers of liver damage, how it affects patients, and whether it could predict the course and outcomes of coronavirus disease 2019 (COVID-19) itself remain unclear.
Although liver injury in patients with COVID-19 is often transient and is usually normalized without special treatment in mild cases of the disease, it can be the first sign of life-threatening events such as acute liver failure in severe and critical cases. Therefore, it is essential for everyday clinical practice to have a more precise view of how the liver impairment affects the course and outcomes of SARS-CoV-2 infection itself. Our study contributes to this goal.
This study aims to analyze the impact of liver enzyme abnormalities on the severity and outcomes of COVID-19 in hospitalized patients to have a clearer view of how to evaluate the risk of severe liver impairment from elevated enzyme tests.
In this study, 684 depersonalized medical records from patients hospitalized with COVID-19 during the 2020-2021 period were analyzed. Patients were assigned to two groups: those with elevated liver enzymes, where at least one out of four liver enzymes were elevated at any point of hospitalization, from admission to discharge; and the control group, with normal liver enzymes during hospitalization. COVID-19 severity was assessed according to the interim World Health Organization guidance (2022). Data on viral pneumonia complications, laboratory tests, and underlying diseases were also collected and analyzed.
In total, 88.2% of patients with SARS-CoV-2 infection produced abnormal liver test results. Alanine aminotransferase and aspartate aminotransferase levels were elevated by a factor of less than 3 in 54.9% and 74.8% of cases with increased enzyme levels, respectively. Patients in Group 1 had almost double the chance of bacterial viral pneumonia complications, required oxygen supply more often, and displayed higher biochemical inflammation indices than those in Group 2. Like in other research, our patients rarely experienced acute liver failure. The majority of the deceased patients had at least one underlying disease or a combination thereof, and most were male. Alongside male gender and older age, diabetes and hyperlipidemia, but not obesity, were confirmed as independent factors associated with more a severe COVID-19 infection in our cohort.
In our study, the presence of liver impairment allows us to predict a more severe inflammation with a higher risk of bacterial complication and worse outcomes of COVID-19. Therefore, monitoring liver enzyme levels should be a part of the qualitative care of patients with SARS-CoV-2 pneumonia.
To find out more precisely the sources of increased liver enzymes in patients with COVID-19, it would be beneficial to elucidate whether the SARS-CoV-2 virus can enter and replicate in hepatocytes. For this purpose, an experimental study on the cell line of the liver origin or virus detection in hepatocytes during a histological analysis of autopsies could be promising.