Published online Jun 14, 2019. doi: 10.3748/wjg.v25.i22.2763
Peer-review started: March 28, 2019
First decision: April 11, 2019
Revised: April 15, 2019
Accepted: May 3, 2019
Article in press: May 3, 2019
Published online: June 14, 2019
Processing time: 79 Days and 0.6 Hours
As one of the most frequent cancers, gastric cancer (GC) caused more than 700000 deaths in just 2012 worldwide. Many recent studies have demonstrated the molecular mechanisms involved in transcriptional regulation in GC, and long noncoding RNAs (lncRNAs) play an irreplaceable role in the initiation and progression of GC, such as maintaining cell growth, evasion of apoptosis, promotion of invasion and metastasis, stemness maintenance, and EMT.
To identify more biomarkers for the diagnosis and treatment of GC.
This study aimed to investigate the underlying mechanisms of HOXA11-AS in GC.
HOXA11-AS expression was detected by qRT-PCR assay in GC tissues, cell lines, and serum samples. Clinicopathological characteristics were collected and expression analysis of HOXA11-AS was performed to evaluate the role of HOXA11-AS. Cell function assays were performed to explore the functions of HOXA11-AS in GC cell lines. Moreover, Western blot was performed to explore the target regulated by HOXA11-AS in GC cell lines.
We found that HOXA11-AS was upregulated in GC tissues, cell lines, and serum samples, and exhibited a significant negative correlation with tumor size, TNM stage, and lymph node metastasis. Cell experiments showed that HOXA11-AS promoted the proliferation and invasion capacity of GC cell lines, and SRSF1 may be the target regulated by HOXA11-AS in GC cells. Especially, GC patients with a lower HOXA11-AS level had a better overall survival rate.
Our study demonstrated that HOXA11-AS can significantly promote GC cell growth, migration, and invasion. Furthermore, it can work through SRSF1. Therefore, our study provides the possible molecular mechanism and two new biomarkers for GC.
In the future, research may reveal the important role of HOXA11-AS that enhances the sensitivity of GC detection and facilitate its application in anti-cancer treatments. The identification of the HOXA11-AS/SRSF1 molecular axis may further explain the underlying mechanism.