Published online Apr 21, 2019. doi: 10.3748/wjg.v25.i15.1890
Peer-review started: December 27, 2018
First decision: January 18, 2019
Revised: February 17, 2019
Accepted: February 22, 2019
Article in press: February 23, 2019
Published online: April 21, 2019
Processing time: 112 Days and 21.1 Hours
Hepatocellular carcinoma (HCC) is a malignant tumor with a high mortality rate. Exosomes have been shown to play an important role in tumorigenesis, cancer development, metastasis, deterioration, and immune escape.
We aimed to research the mechanism of exosomal microRNA-224 (miR-224) and its target in the development and invasion of HCC, and we also evaluated the diagnostic and prognostic value of miR-224 for patients with HCC.
Cell culture and transfection of exosomal miR-224, its mimics, and its inhibitor; real-time quantitative PCR; and luciferase reporter assay were used to explore the mechanism of exosomal miR-224. HCC patients and healthy controls were used to assess the value of exosomal miR-224 in diagnosing HCC and predicting HCC prognosis.
Serum exosomes incubated with the miR-224 mimic showed a significant increase in cell proliferation and invasion when compared to the control group, while those incubated with the inhibitor showed a significant reduction. For discriminating HCC from healthy controls, serum exosomal miR-224 showed an area under the ROC curve of 0.910. Higher serum exosomal miR-224 expression levels in HCC patients were associated with lower overall survival.
The results showed that exosomal miR-224 directly targets the 3'-UTR of glycine N-methyltransferaseto and impacts the proliferation and invasion of HCC, and exosomal miR-224 may be used as a potential diagnostic and prognostic factor for patients with HCC.
Our study provides novel insight into the mechanism of exosomal miR-224 in the development and invasion of HCC and may provide a potential biomarker for the diagnosis and prognosis of HCC.