Basic Research
Copyright ©The Author(s) 2003. Published by Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Jun 15, 2003; 9(6): 1273-1277
Published online Jun 15, 2003. doi: 10.3748/wjg.v9.i6.1273
Expression and cell-specific localization of cholecystokinin receptors in rat lung
Bin Cong, Shu-Jin Li, Yi-Ling Ling, Yu-Xia Yao, Zhen-Yong Gu, Jun-Xia Wang, Hong-Yu You
Bin Cong, Shu-Jin Li, Yi-Ling Ling, Yu-Xia Yao, Zhen-Yong Gu, Department of Pathophysiology, Hebei Medical University, Shijiazhuang 050017, Hebei Province, China
Jun-Xia Wang, Hong-Yu You, Molecular Biological Laboratory, Hebei Medical University, Shijiazhuang 050017, Hebei Province, China
Author contributions: All authors contributed equally to the work.
Supported by National Natural Science Foundation of China, No. 39570304
Correspondence to: Professor Yi-Ling Ling, Department of Pathophysiology, Hebei Medical University, Shijiazhuang 050017, Hebei Province, China. lingyiling@163.net
Telephone: +86-311-6265645
Received: January 4, 2003
Revised: February 4, 2003
Accepted: February 11, 2003
Published online: June 15, 2003
Abstract

AIM: To elucidate whether CCK receptors exist in lung tissues and their precise cellular localization in the lung.

METHODS: CCK-AR and CCK-BR mRNA expression and cellular distribution in the rat lung were detected by highly sensitive method of in situ reverse transcription-polymerase chain reaction (RT-PCR) and conventional in situ hybridization.

RESULTS: CCK-AR and CCK-BR gene positive signals were observed in bronchial epithelial cells, alveolar epithelial cells, pulmonary macrophages and vascular endothelial cells of the rats lung by in situ RT-PCR. The hybridization signals of CCK-AR were relatively faint. By in situ hybridization, however, only the signals of CCK-BR but not CCK-AR were detected in the lung, and the positive staining was only found in vascular endothelial cells and macrophages.

CONCLUSION: CCK-AR and CCK-BR gene were present in pulmonary vascular endothelial cells, macrophages, bronchial epithelial cells and alveolar epithelial cells, which play an important role in mediating the regulatory actions of CCK-8 on these cells.

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