Viral Liver Diseases
Copyright ©The Author(s) 2002. Published by Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Jun 15, 2002; 8(3): 499-504
Published online Jun 15, 2002. doi: 10.3748/wjg.v8.i3.499
Full-length core sequence dependent complex-type glycosylation of hepatitis C virus E2 glycoprotein
Li-Xin Zhu, Jing Liu, Ying-Chun Li, Yu-Ying Kong, Caroline Staib, Gerd Sutter, Yuan Wang, Guang-Di Li
Li-Xin Zhu, Jing Liu, Ying-Chun Li, Yu-Ying Kong, Yuan Wang, Guang-Di Li, Institute of Biochemistry and Cell Biology, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, Shanghai 200031, China
Caroline Staib, Gerd Sutter, GSF-Institut für Molekulare Virologie, Trogerstr. 4b, 81675 München, Germany.
Author contributions: The first two authors contributed equally to this paper.
Supported by the National 863 High Technology Foundation of China, No. 863-102-07-02-02, No. 2001AA215171 and the project CHN 98/112 (WTZ-Internationales Büro des BMBF).
Correspondence to: Yuan Wang and Guang-Di Li, Institute of Biochemistry and Cell Biology, Shanghai Institutes for Biological Sciences, Chinese Academy of Sciences, 320 Yue-Yang Road, Shanghai 200031, China. wangyuan@server.shcnc.ac.cn
Telephone: +86-21-64374430 Fax: +86-21-64338357
Received: December 5, 2001
Revised: December 23, 2001
Accepted: January 23, 2002
Published online: June 15, 2002
Abstract

AIM: To study HCV polyprotein processing is important for the understanding of the natural history of HCV and the design of vaccines against HCV. The purpose of this study is to investigate the affection of context sequences on hepatitis C virus (HCV) E2 processing.

METHODS: HCV genes of different lengths were expressed and compared in vaccinia virus/T7 system with homologous patient serum S94 and mouse anti-serum ME2116 raised against E. coli-derived E2 peptide, respectively. Deglycosylation analysis and GNA (Galanthus nivalus) lectin binding assay were performed to study the post-translational processing of the expressed products.

RESULTS: E2 glycoproteins with different molecular weights (~75 kDa and ~60 kDa) were detected using S94 and ME2116, respectively. Deglycosylation analysis showed that this difference was mainly due to different glycosylation. Endo H resistance and its failure to bind to GNA lectin demonstrated that the higher molecular weight form (75 kDa) of E2 was complex-type glycosylated, which was readily recognized by homologous patient serum S94. Expression of complex-type glycosylated E2 could not be detected in all of the core-truncated constructs tested, but readily detected in constructs encoding full-length core sequences.

CONCLUSION: The upstream conserved full-length core coding sequence was required for the production of E2 glycoproteins carrying complex-type N-glycans which reacted strongly with homologous patient serum and therefore possibly represented more mature forms of E2. As complex-type N-glycans indicated modification by Golgi enzymes, the results suggest that the presence of full-length core might be critical for E1/E2 complex to leave ER. Our data may contribute to a better understanding of the processing of HCV structural proteins as well as HCV morphogenesis.

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