Liver Cancer
Copyright ©The Author(s) 2002. Published by Baishideng Publishing Group Inc. All rights reserved.
World J Gastroenterol. Jun 15, 2002; 8(3): 464-468
Published online Jun 15, 2002. doi: 10.3748/wjg.v8.i3.464
Antitumor activities of human autologous cytokine-induced killer (CIK) cells against hepatocellular carcinoma cells in vitro and in vivo
Fu-Sheng Wang, Ming-Xu Liu, Bing Zhang, Ming Shi, Zhou-Yun Lei, Wen-Bing Sun, Qing-You Du, Ju-Mei Chen
Fu-Sheng Wang, Ming-Xu Liu, Bing Zhang, Ming Shi, Zhou-Yun Lei, Wen-Bing Sun, Qing-You Du, Ju-Mei Chen, Division of Biological Engineering, Beijing Institute of Infectious Diseases, Beijing 100039, China
Wen-Bing Sun, Department of Surgery, Beijing Hospital of Infectious Diseases, Beijing 100039, China
Author contributions: All authors contributed equally to the work.
Supported by Science and Technology Development Foundation of Beijing Institute of Infectious Diseases, No. 01Z094
Correspondence to: Dr. Fu-Sheng Wang, Division of Biological Engineering, Beijing Institute of Infectious Diseases, 26 Fengtai Road, Beijing 100039, China. fswang@public.bta.net.cn
Telephone: +86-10-66933332 Fax: +86-10-63831870
Received: April 11, 2001
Revised: April 23, 2001
Accepted: February 25, 2002
Published online: June 15, 2002
Abstract

AIM: To characterize the anticancer function of cytokine-induced killer cells (CIK) and develop an adoptive immunotherapy for the patients with primary hepatocellular carcinoma (HCC), we evaluated the proliferation rate, phenotype and the antitumor activity of human CIK cells from healthy donors and HCC patients in vitro and in vivo.

METHODS: Peripheral blood mononuclear cells (PBMC) from healthy donors and patients with primary HCC were incubated in vitro and induced into CIK cells in the presence of various cytokines such as interferon-gamma (IFN-γ), interleukin-1 (IL-1), IL-2, and monoclonal antibody (mAb) against CD3. The phenotype and characterization of CIK cells were identified by flow cytometric analysis. The cytotoxicity of CIK cells was determined by 51Cr release assay.

RESULTS: The CIK cells were shown to be a heterogeneous population with different cellular phenotypes. The percentage of CD3+CD56+ positive cells, the dominant effector cells, in total CIK cells from healthy donors and HCC patients, significantly increased from 0.1%-0.13% at day 0 to 19.0%-20.5% at day 21 incubation, which suggested that the CD3+CD56+ positive cells proliferated faster than other cell populations of CIK cells in the protocol used in this study. After 28 day in vitro incubation, the CIK cells from patients with HCC and healthy donors increased by more than 300-fold and 500-fold in proliferation cell number, respectively. CIK cells originated from HCC patients possessed a higher in vitro antitumor cytotoxic activity on autologous HCC cells than the autologous lymphokine-activated killer (LAK) cells and PBMC cells. In in vivo animal experiment, CIK cells had stronger effects on the inhibition of tumor growth in Balb/c nude mice bearing BEL-7402-producing tumor than LAK cells (mean inhibitory rate, 84.7% vs 52.8%, P < 0.05) or PBMC (mean inhibitory rate, 84.7% vs 37.1%, P < 0.01).

CONCLUSION: Autologous CIK cells are of highly efficient cytotoxic effector cells against primary hepatocellular carcinoma cells and might serve as an alternative adoptive therapeutic strategy for HCC patients.

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