Published online Nov 7, 2024. doi: 10.3748/wjg.v30.i41.4496
Revised: September 12, 2024
Accepted: September 25, 2024
Published online: November 7, 2024
Processing time: 78 Days and 21.7 Hours
In this manuscript, we comment on the article, which explores the anti-cancer effects of Calculus bovis (CB) in tumor biology. We highlight its potential, particularly in hepatocellular carcinoma (HCC), where it inhibits the phosphatidylinositol 3-kinase/protein kinase B/mammalian target of rapamycin pathways and induces apoptosis. CB contains compounds such as oleanolic acid and ursolic acid that target interleukin-6, mitogen-activated protein kinase 8, vascular endothelial growth factor, and caspase-3, offering anti-inflammatory and hepatoprotective benefits. The manuscript also discusses CB sativus (CBS), an artificial substitute, which has shown efficacy in reducing hepatic inflammation and oxidative stress in animal models. We emphasize the need for further research on the effects of CBS on the gut-liver axis and gut microbiota, and on targeting Wnt signaling and M2 tumor-associated macrophage as potential therapeutic strategies against HCC.
Core Tip: The traditional Chinese medicine Calculus bovis (CB) is known to affect tumor biology by showing promise in inhibiting hepatocellular carcinoma (HCC) progression by targeting the Wnt signaling pathway. The study highlights CB's anti-cancer properties, particularly against liver cancer, through modulation of pathways such as phosphatidylinositol 3-kinase/protein kinase B/mammalian target of rapamycin, and compounds such as oleanolic acid and ursolic acid. Additionally, CB sativus has shown efficacy in reducing hepatic inflammation and oxidative stress, suggesting potential therapeutic strategies for HCC, warranting further clinical validation.