Published online Nov 14, 2022. doi: 10.3748/wjg.v28.i42.6034
Peer-review started: August 27, 2022
First decision: September 30, 2022
Revised: October 6, 2022
Accepted: November 2, 2022
Article in press: November 2, 2022
Published online: November 14, 2022
Processing time: 74 Days and 19 Hours
The disease burden related to hepatocellular carcinoma (HCC) is increasing. Most HCC patients are diagnosed at the advanced stage and multikinase inhibitors have been the only treatment choice for them. Recently, the approval of immune checkpoint inhibitors (ICIs) has provided a new therapeutic strategy for HCC. It is noteworthy that the positive outcomes of the phase III clinical trial IMBrave150 [atezolizumab (anti-programmed cell death ligand 1 antibody) combined with bevacizumab (anti-vascular endothelial growth factor monoclonal antibody)], showed that overall survival and progression-free survival were significantly better with sorafenib. This combination therapy has become the new standard therapy for advanced HCC and has also attracted more attention in the treatment of HCC with anti-angiogenesis-immune combination therapy. Currently, the synergistic antitumor efficacy of this combination has been shown in many preclinical and clinical studies. In this review, we discuss the mechanism and clinical application of anti-angiogenics and immunotherapy in HCC, outline the relevant mechanism and rationality of the combined application of anti-angiogenics and ICIs, and point out the existing challenges of the combination therapy.
Core Tip: Anti-angiogenesis combined with immunotherapy in hepatocellular carcinoma (HCC) has attracted extensive attention. Although the mechanism of these combinations is largely understood, the biomarkers for predicting treatment response are still lacking. Thus, we outline the relevant mechanism and rationality of the combined application of anti-angiogenics and immune checkpoint inhibitors, and further explore the biomarkers that are associated with treatment response, which are critical in studies of HCC.