Published online Jun 7, 2021. doi: 10.3748/wjg.v27.i21.2727
Peer-review started: January 20, 2021
First decision: February 9, 2021
Revised: February 23, 2021
Accepted: April 13, 2021
Article in press: April 13, 2021
Published online: June 7, 2021
Processing time: 126 Days and 23.6 Hours
Hepatitis B virus (HBV) infection, although preventable by vaccination, remains a global health problem and a major cause of chronic liver disease. Although current treatment strategies suppress viral replication very efficiently, the optimal endpoint of hepatitis B surface antigen (HBsAg) clearance is rarely achieved. Moreover, the thorny problems of persistent chromatin-like covalently closed circular DNA and the presence of integrated HBV DNA in the host genome are ignored. Therefore, the scientific community has focused on developing innovative therapeutic approaches to achieve a functional cure of HBV, defined as undetectable HBV DNA and HBsAg loss over a limited treatment period. A deeper understanding of the HBV life cycle has led to the introduction of novel direct-acting antivirals that exert their function through multiple mechanisms, including inhibition of viral entry, transcriptional silencing, epigenetic manipulation, interference with capsid assembly, and disruption of HBsAg release. In parallel, another category of new drugs aims to restore dysregulated immune function in chronic hepatitis B accompanied by lethargic cellular and humoral responses. Stimulation of innate immunity by pattern-recognition receptor agonists leads to upregulation of antiviral cytokine expression and appears to contribute to HBV containment. Immune checkpoint inhibitors and adoptive transfer of genetically engineered T cells are breakthrough technologies currently being explored that may elicit potent HBV-specific T-cell responses. In addition, several clinical trials are attempting to clarify the role of therapeutic vaccination in this setting. Ultimately, it is increasingly recognized that elimination of HBV requires a treatment regimen based on a combination of multiple drugs. This review describes the rationale for progressive therapeutic interventions and discusses the latest findings in the field of HBV therapeutics.
Core Tip: Despite preventive vaccination and effective antiviral drugs, approximately 300 million people worldwide are at risk of liver-related morbidity and mortality from hepatitis B virus (HBV) infection. In the search for more effective strategies, research has focused on two broad categories of therapeutic intervention. First, direct-acting antivirals that interrupt various stages of the HBV life cycle are yielding promising results in clinical trials that show suppression of the HBV antigen load. Second, the need to address depleted immune responses and revive the tolerogenic liver microenvironment has brought immunotherapies to the forefront. It is likely that an upcoming treatment that combines agents from both classes will achieve HBV elimination.