Published online May 7, 2019. doi: 10.3748/wjg.v25.i17.2019
Peer-review started: February 22, 2019
First decision: March 20, 2019
Revised: March 25, 2019
Accepted: March 29, 2019
Article in press: March 30, 2019
Published online: May 7, 2019
Processing time: 74 Days and 17.8 Hours
The prevalence of non-alcoholic fatty liver disease (NAFLD) is rising exponentially worldwide. The spectrum of NAFLD includes non-alcoholic fatty liver, non-alcoholic steatohepatitis, liver cirrhosis, and even hepatocellular carcinoma. Evidence shows that microbial metabolites play pivotal roles in the onset and progression of NAFLD. In this review, we discuss how microbe-derived metabolites, such as short-chain fatty acids, endogenous ethanol, bile acids and so forth, contribute to the pathogenesis of NAFLD.
Core tip: Non-alcoholic fatty liver disease (NAFLD) is a global epidemic metabolic disease lacking effective therapeutic strategies and the internal pathogenesis is still uncertain. Gut microbiota-derived metabolites have attracted much attention for its association with the onset and progression of NAFLD. In this review, we mainly elucidate the diverse roles of microbe-derived metabolites in the development of NAFLD, which is conducive to better understanding the biological functions of microbial metabolites in NAFLD via the gut-liver axis and facilitating the excavation of potential therapeutic approaches for NAFLD.