Published online Mar 28, 2018. doi: 10.3748/wjg.v24.i12.1285
Peer-review started: January 10, 2018
First decision: February 5, 2018
Revised: February 18, 2018
Accepted: March 7, 2018
Article in press: March 7, 2018
Published online: March 28, 2018
Processing time: 75 Days and 13.7 Hours
Hepatopulmonary syndrome (HPS) is a frequent pulmonary complication of end-stage liver disease, characterized by impaired arterial oxygenation induced by intrapulmonary vascular dilatation. Its prevalence ranges from 4% to 47% in patients with cirrhosis due to the different diagnostic criteria applied among different studies. Nitric oxide overproduction and angiogenesis seem to be the hallmarks of a complicated pathogenetic mechanism, leading to intrapulmonary shunting and ventilation-perfusion mismatch. A classification of HPS according to the severity of hypoxemia has been suggested. Contrast-enhanced echocardiography represents the gold standard method for the detection of intrapulmonary vascular dilatations which is required, in combination with an elevated alveolar arterial gradient to set the diagnosis. The only effective treatment which can modify the syndrome’s natural history is liver transplantation. Although it is usually asymptomatic, HPS imparts a high risk of pretransplantation mortality, independently of the severity of liver disease, while there is variable data concerning survival rates after liver transplantation. The potential of myocardial involvement in the setting of HPS has also gained increasing interest in recent research. The aim of this review is to critically approach the existing literature of HPS and emphasize unclear points that remain to be unraveled by future research.
Core tip: Hepatopulmonary syndrome (HPS) constitutes a relatively frequent complication of end-stage liver disease, characterized by impairment of arterial oxygenation. The only effective treatment is liver transplantation, improving hypoxemia. While there are controversial data regarding HPS prognosis before and after liver transplantation, the question remains whether HPS constitutes an independent factor of morbidity, providing HPS patients priority for liver transplantation. Furthermore, possible associations with myocardial function, which could support the utility of echocardiographical parameters as markers of HPS, remain yet to be established.