Published online Jan 7, 2018. doi: 10.3748/wjg.v24.i1.15
Peer-review started: November 3, 2017
First decision: November 14, 2017
Revised: November 23, 2017
Accepted: November 27, 2017
Article in press: November 27, 2017
Published online: January 7, 2018
Processing time: 65 Days and 13.5 Hours
Although the incidence of Crohn’s disease (CD) in China is not as high as that in European and American countries, there has been a clear increasing trend in recent years. Little is known about its pathogenesis, cause of deferment, and the range of complications associated with the disease. Local and international scholars have presented many hypotheses about CD pathogenesis based on experimental and clinical studies, including genetic susceptibility, immune function defects, intestinal microflora disorders, delayed hypersensitivity, and food antigen stimulation. However, the specific mechanism leading to this immune imbalance, which causes persistent intestinal mucosal damage, and the source of the inflammatory cascade reaction are still unclear. So far, the results of research studies differ locally and internationally. This paper presents the most current research on immune factors in the pathogenesis of CD.
Core tip: It is now clear that Crohn's disease (CD) is an autoimmune disease that involves at least the intestinal mucosal immune system, when the mucosal immune system is invaded by food or bacterial antigens. However, it is worth mentioning that the mechanism remains unclear. Whether activation of the immune system is the internal defects (constitutive activation or regulation mechanism disorder) or changes in the epithelial mucosal barrier leading to continuous stimulation is still not clear. The mechanism of CD is intensively studied by domestic and foreign scholars on the immune destruction. Here we summarize the latest research status on immune factors in the pathogenesis of CD with regard to the mechanisms about T-lymphocyte immunity, innate lymphocyte immunity, cytokines and immune therapy.