Published online May 7, 2017. doi: 10.3748/wjg.v23.i17.3066
Peer-review started: September 12, 2016
First decision: October 20, 2016
Revised: December 7, 2016
Accepted: January 18, 2017
Article in press: January 18, 2017
Published online: May 7, 2017
Processing time: 236 Days and 22.8 Hours
To comparatively investigate the cellular and molecular characteristics of low-grade slightly elevated adenomas and polypoid adenomas.
Colorectal tumors were collected from 24 patients with slightly elevated adenomas and 23 patients with polypoid adenomas. Five commonly mutated genes (APC, BRAF, KRAS, NRAS, and PIK3CA) were selected for mutational analysis. Paraffin-embedded tumor sections were used to calculate the apoptotic index (AI) and Ki-67 labeling index (KLI). Two pure colorectal epithelial cell lines were created by pooling the slightly elevated and polypoid tumors. Western blots, luciferase assays for β-catenin-T-cell factor protein/β-catenin-lymphoid enhancer factor (β-catenin-TCF/LEF)-driven transcriptional activity, and caspase activity assays were conducted on the two cell lines.
Slightly elevated lesions showed a significantly lower APC mutational frequency and a significantly higher KRAS mutational frequency (both P < 0.05). Slightly elevated lesions showed a significantly lower AI (P < 0.05). β-catenin and β-catenin-TCF/LEF-driven transcriptional activity was significantly upregulated in slightly elevated lesions (both P < 0.05). In slightly elevated lesions, c-Myc was significantly downregulated, while cyclin D1 was significantly upregulated (both P < 0.05). β-catenin-TCF/LEF-driven transcriptional activity was negatively correlated with c-Myc (ρ = -0.78). Slightly elevated lesions displayed significant Bcl-2 and Bcl-xL upregulation (both P < 0.05) along with significant decreases in caspase-9 and caspase-3 activity (both P < 0.05). c-Myc was negatively correlated with Bcl-2 and Bcl-xL (ρ = -0.74 and -0.78, respectively).
The lower apoptotic activity of low-grade slightly elevated adenomas can be partly attributed to upregulated β-catenin pathway activity and downregulated c-Myc expression.
Core tip: We comparatively investigated the cellular and molecular characteristics of low-grade slightly elevated adenomas and low-grade polypoid adenomas. Slightly elevated lesions displayed a significantly lower apoptotic index as well as significantly upregulated β-catenin expression and β-catenin-TCF/LEF-driven transcriptional activity. Slightly elevated lesions also displayed c-Myc downregulation, cyclin D1 upregulation, upregulation of Bcl-2 and Bcl-xL, and downregulation of caspase-9 and caspase-3 activity. Low-grade slightly elevated adenomas displayed a lower apoptotic activity relative to low-grade polypoid adenomas, which can be partly attributed to upregulated β-catenin pathway activity and downregulated c-Myc expression.