Published online Dec 21, 2015. doi: 10.3748/wjg.v21.i47.13259
Peer-review started: August 1, 2015
First decision: August 26, 2015
Revised: August 31, 2015
Accepted: October 17, 2015
Article in press: October 20, 2015
Published online: December 21, 2015
Processing time: 137 Days and 2 Hours
AIM: To evaluate the epithelial-to-mesenchymal transition (EMT) of circulating tumor cells (CTCs) in gastric cancer patients.
METHODS: We detected tumor cells for expression of four epithelial (E+) transcripts (keratins 8, 18, and 19 and epithelial cell adhesion molecule) and two mesenchymal (M+) transcripts (Vimentin and Twist) by a quantifiable, dual-colorimetric RNA-in situ hybridization assay. Between July 2014 and October 2014, 44 patients with gastric cancer were recruited for CTC evaluation. Blood samples were obtained from selected patients during the treatment course [before surgery, after surgery and at the 6th cycle of XELOX based chemotherapy (about 6 mo postoperatively)].
RESULTS: We found the EMT phenomenon in which there were a few biphenotypic E+/M+ cells in primary human gastric cancer specimens. Of the 44 patients, the presence of CTCs was reported in 35 (79.5%) patients at baseline. Five types of cells including from exclusively E+ CTCs to intermediate CTCs and exclusively M+ CTCs were identified (4 patients with M+ CTCs and 10 patients with M+ or M+ > E+ CTCs). Further, a chemotherapy patient having progressive disease showed a proportional increase of mesenchymal CTCs in the post-treatment blood specimens. We used NCI-N87 cells to analyze the linearity and sensitivity of CanPatrolTM system and the correlation coefficient (R2) was 0.999.
CONCLUSION: The findings suggest that the EMT phenomenon was both in a few cells of primary tumors and abundantly in CTCs from the blood of gastric cancer patients, which might be used to monitor therapy response.
Core tip: Epithelial-to-mesenchymal transition has been thought to play a critical role in tumor metastatic progression in preclinical models, however, characterizing the epithelial vs mesenchymal phenotypes of circulating tumor cells has been challenging. In this study, we aimed to evaluate epithelial-to-mesenchymal transition phenomenon in circulating gastric tumor cells by a combination of physical and biological methods. Our findings have provided evidence of the phenomenon both in rare cells within primary tumors and more abundantly in circulating tumor cells. Furthermore, we demonstrated that the evaluation of the mesenchymal circulating tumor cells in peripheral blood can be used to monitor therapy response in gastric cancer patients.