Published online Jun 7, 2015. doi: 10.3748/wjg.v21.i21.6550
Peer-review started: November 24, 2014
First decision: January 8, 2015
Revised: February 10, 2015
Accepted: March 12, 2015
Article in press: March 12, 2015
Published online: June 7, 2015
Processing time: 199 Days and 15.8 Hours
AIM: To investigate alternative splicing in vascular endothelial growth factor A (VEGFA), amyloid beta precursor protein (APP), and Numb homolog (NUMB) in colorectal cancer (CRC).
METHODS: Real-time quantitative reverse transcriptase polymerase chain reaction (qRT-PCR) and PCR-restriction fragment length polymorphism analyses were performed to detect the expression of VEGFA, APP, and NUMB mRNA in 20 CRC tissues and matched adjacent normal tissues, as well as their alternative splicing variants.
RESULTS: qRT-PCR analysis revealed that the expression of APP, NUMB, and VEGFA165b mRNA were significantly downregulated, while VEGFA mRNA was upregulated, in CRC tissues (all P < 0.05). PCR-restriction fragment length polymorphism analysis revealed that the expression of VEGFA165a/b in CRC tissues was significantly higher than in adjacent normal tissues (P < 0.05). Compared with adjacent normal tissues, the expression of NUMB-PRRS in CRC tissues was significantly decreased (P < 0.05), and the expression of NUMB-PRRL was increased (P < 0.05).
CONCLUSION: Alternative splicing of VEGFA, APP, and NUMB may regulate the development of CRC, and represent new targets for its diagnosis, prognosis, and treatment.
Core tip: This study was undertaken to investigate the effect of alternative splicing of vascular endothelial growth factor A (VEGFA), amyloid beta precursor protein (APP), and Numb homolog (NUMB) genes in colorectal cancer (CRC). We demonstrated that these genes and their alternative splice variants were different in CRC tissues and matched adjacent normal tissues using quantitative reverse transcriptase PCR and PCR-restriction fragment length polymorphism analyses. We conclude that alternative splicing of VEGFA, APP, and NUMB may be associated with the diagnosis, prognosis, and treatment of CRC. This study may help in understanding the relationship between alternative splicing and CRC.