Published online Jan 7, 2015. doi: 10.3748/wjg.v21.i1.301
Peer-review started: May 13, 2014
First decision: June 10, 2014
Revised: June 24, 2014
Accepted: July 29, 2014
Article in press: July 30, 2014
Published online: January 7, 2015
Processing time: 239 Days and 16 Hours
AIM: To investigate whether liver steatosis reduction due to a six-month dietary intervention results in significant changes in the concentrations of fatty acids.
METHODS: A group of 35 Caucasian individuals diagnosed with different levels of steatosis were prospectively enrolled in the present study. Analysis of the fatty acid profiles was performed according to changes in liver steatosis (liver steatosis reduction by one or two degrees) after a six-month dietary intervention. The diet helped reduce body mass in obese and overweight patients, and stabilize both glycemia and dyslipidemia. Fatty acids were extracted according to the Folch method and analyzed by gas chromatography.
RESULTS: This study showed significant changes in fatty acid profiles in patients who had reduced liver steatosis by one as well as two degrees. A reduction in liver steatosis by one degree caused a significant increase in the level of the n-3 family: eicosapentaenoic acid (P < 0.055), docosapentaenoic acid-C 22:5 (P < 0.05) and docosahexaenoic acid (P < 0.05). A reduction in liver steatosis by two degrees caused a significant decrease in serum palmitoleic acid-C 16:1 (P < 0.05).
CONCLUSION: Liver steatosis reduction is associated with changes in fatty acid profiles, and these changes may reflect an alteration in fatty acid synthesis and metabolism. These findings may help better understand regression of nonalcoholic fatty liver disease.
Core tip: A reduction in liver steatosis due to a six-month dietary intervention resulted in significant changes in the concentrations of fatty acids. These changes may reflect an alteration in fatty acid synthesis and metabolism. These results show key elements in the mechanism of the reduction of liver steatosis and allow for a better understanding of the regression of nonalcoholic fatty liver disease.