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World J Gastroenterol. Jul 7, 2014; 20(25): 8005-8010
Published online Jul 7, 2014. doi: 10.3748/wjg.v20.i25.8005
Peculiar characteristics of portal-hepatic hemodynamics of alcoholic cirrhosis
Massimo Bolognesi, Alberto Verardo, Marco Di Pascoli
Massimo Bolognesi, Alberto Verardo, Marco Di Pascoli, Department of Medicine, University of Padua, Azienda Ospedaliera Università di Padova, 35128 Padova, Italy
Author contributions: Bolognesi M, Verardo A and Di Pascoli M have given substantial contributions to conception and design of the review, for drafting the article and revising it; all authors have given final approval of the version to be published.
Correspondence to: Massimo Bolognesi, MD, PhD, Department of Medicine, University of Padua, Azienda Ospedaliera Università di Padova, Clinica Medica 5, Via Giustiniani 2, 35128 Padova, Italy. massimo.bolognesi@unipd.it
Telephone: +39-49-8212383 Fax: +39-49-8754179
Received: October 24, 2013
Revised: December 31, 2013
Accepted: February 26, 2014
Published online: July 7, 2014
Processing time: 251 Days and 21.1 Hours
Abstract

Alcohol-related cirrhosis is a consequence of heavy and prolonged drinking. Similarly to patients with cirrhosis of other etiologies, patients with alcoholic cirrhosis develop portal hypertension and the hepatic, splanchnic and systemic hemodynamic alterations that follow. However, in alcoholic cirrhosis, some specific features can be observed. Compared to viral cirrhosis, in alcohol-related cirrhosis sinusoidal pressure is generally higher, hepatic venous pressure gradient reflects portal pressure better, the portal flow perfusing the liver is reduced despite an increase in liver weight, the prevalence of reversal portal blood flow is higher, a patent paraumbilical vein is a more common finding and signs of hyperdynamic circulations, such as an increased cardiac output and decreased systemic vascular resistance, are more pronounced. Moreover, alcohol consumption can acutely increase portal pressure and portal-collateral blood flow. Alcoholic cardiomyopathy, another pathological consequence of prolonged alcohol misuse, may contribute to the hemodynamic changes occurring in alcohol-related cirrhosis. The aim of this review was to assess the portal-hepatic changes that occur in alcohol-related cirrhosis, focusing on the differences observed in comparison with patients with viral cirrhosis. The knowledge of the specific characteristics of this pathological condition can be helpful in the management of portal hypertension and its complications in patients with alcohol-related cirrhosis.

Keywords: Alcohol-related cirrhosis; Portal hypertension; Splanchnic flow; Hyperdynamic circulatory syndrome; Alcohol-related cardiomyopathy

Core tip: Patients with alcoholic cirrhosis develop portal hypertension and hemodynamics alterations similarly to patients with cirrhosis of other etiologies, but some specific features can be observed. Compared to viral cirrhosis, sinusoidal pressure is generally higher, hepatic venous pressure gradient reflects portal pressure better, the portal flow perfusing the liver is reduced despite an increase in liver weight, the prevalence of reversal portal blood flow is higher, a patent paraumbilical vein is a more common finding and signs of hyperdynamic circulations, such as an increased cardiac output and decreased systemic vascular resistance, are more pronounced.