Published online Jun 21, 2014. doi: 10.3748/wjg.v20.i23.7325
Revised: December 24, 2013
Accepted: January 8, 2014
Published online: June 21, 2014
Processing time: 251 Days and 14.5 Hours
Autophagy is a mechanism involved in cellular homeostasis under basal and stressed conditions delivering cytoplasmic content to the lysosomes for degradation to macronutrients. The potential role of autophagy in disease is increasingly recognised and investigated in the last decade. Nowadays it is commonly accepted that autophagy plays a role in the hepatic lipid metabolism. Hence, dysfunction of autophagy may be an underlying cause of non-alcoholic fatty liver disease. However, controversy of the exact role of autophagy in the lipid metabolism exists: some publications report a lipolytic function of autophagy, whereas others claim a lipogenic function. This review aims to give an update of the present knowledge on autophagy in the hepatic lipid metabolism, hepatic insulin resistance, steatohepatitis and hepatic fibrogenesis.
Core tip: Autophagy is a mechanism involved in cellular homeostasis. In this review the current knowledge on the role of autophagy in non-alcoholic fatty liver disease (NAFLD) is summarised, with emphasis on the current controversy on the lipolytic vs lipogenic function in hepatic lipid metabolism. Furthermore the role of autophagy in the pathophysiology of insulin resistance, hepatocellular injury and fibrogenesis is reviewed to better understand its importance in NAFLD.