Brief Article
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World J Gastroenterol. Sep 7, 2011; 17(33): 3836-3841
Published online Sep 7, 2011. doi: 10.3748/wjg.v17.i33.3836
Ghrelin attenuates gastrointestinal epithelial damage induced by doxorubicin
Mohamed A Fahim, Hazem Kataya, Rkia El-Kharrag, Dena AM Amer, Basel al-Ramadi, Sherif M Karam
Mohamed A Fahim, Department of Physiology, Faculty of Medicine and Health Sciences, UAE University, Al Ain, PO Box 17666, United Arab Emirates
Hazem Kataya, Department of Biology, Faculty of Science, UAE University, Al Ain, PO Box 17666, United Arab Emirates
Rkia El-Kharrag, Sherif M Karam, Department of Anatomy, Faculty of Medicine and Health Sciences, UAE University, Al Ain, PO Box 17666, United Arab Emirates
Rkia El-Kharrag, Dena AM Amer, Basel al-Ramadi, Department of Microbiology and Immunology, Faculty of Medicine and Health Sciences, UAE University, Al Ain, PO Box 17666, United Arab Emirates
Author contributions: Kataya H, Fahim MA, al-Ramadi B and Karam SM designed this study; El-Kharrag R and Amer DAM performed the research; al-Ramadi B and Karam SM contributed analytical tools; El-Kharrag R, Amer DAM, al-Ramadi B and Karam SM analyzed the data; Fahim MA, al-Ramadi B and Karam SM wrote the paper.
Correspondence to: Sherif M Karam, Professor, Department of Anatomy, Faculty of Medicine and Health Sciences, UAE University, Al-Ain, PO Box 17666, United Arab Emirates. skaram@uaeu.ac.ae
Telephone: +971-3-7137493 Fax: +971-3-7672033
Received: December 13, 2010
Revised: March 5, 2011
Accepted: March 12, 2011
Published online: September 7, 2011
Abstract

AIM: To examine the influence of ghrelin on the regenerative potential of gastrointestinal (GI) epithelium.

METHODS: Damage to GI epithelium was induced in mice by two intravenous injections of doxorubicin (10 and 6 mg/kg). Some of the doxorubicin-treated mice received a continuous subcutaneous infusion of ghrelin (1.25 μg/h) for 10 d via implanted mini-osmotic pumps. To label dividing stem cells in the S-phase of the cell cycle, all mice received a single intraperitoneal injection of 5’-bromo-2’-deoxyuridine (BrdU) one hour before sacrifice. The stomach along with the duodenum were then removed and processed for histological examination and immunohistochemistry using anti-BrdU antibody.

RESULTS: The results showed dramatic damage to the GI epithelium 3 d after administration of chemotherapy which began to recover by day 10. In ghrelin-treated mice, attenuation of GI mucosal damage was evident in the tissues examined post-chemotherapy. Immunohistochemical analysis showed an increase in the number of BrdU-labeled cells and an alteration in their distribution along the epithelial lining in response to damage by doxorubicin. In mice treated with both doxorubicin and ghrelin, the number of BrdU-labeled cells was reduced when compared with mice treated with doxorubicin alone.

CONCLUSION: The present study suggests that ghrelin enhances the regenerative potential of the GI epithelium in doxorubicin-treated mice, at least in part, by modulating cell proliferation.

Keywords: Gastrointestinal cell proliferation; Gastro-intestinal mucosal damage; Ghrelin